{"title":"噩梦是应激性心肌病的诱因","authors":"Marc Arcens, Stephane Noble","doi":"10.5603/cj.97167","DOIUrl":null,"url":null,"abstract":"A 67-year-old female was admitted to the emergency department with moderate dyspnea that escalated into oppressive chest pain after 4 hours. She had mild hypertension (153/107 mmHg) and sinus tachycardia (110 bpm). The electrocardiogram showed a left axis deviation without any signs of ischemia and a corrected QT interval of 471 ms (Fig. 1A). The biological markers showed a N-teminal-pro-B-type natriuretic peptide level of 1513 ng/L, peak troponins of 3502 ng/L, and peak creatine kinase of 306 U/L. The transthoracic echocardiogram (TTE) showed severe apical dysfunction with a left ventricular ejection fraction (LVEF) of 25% (Fig. 1B, Suppl. Video 1, Part 1 ). A left ventriculography (Fig. 1C, Suppl. Video 1, Part 2 ) and coronary angiography (Fig. 1D, E)","PeriodicalId":93923,"journal":{"name":"Cardiology journal","volume":null,"pages":null},"PeriodicalIF":0.0000,"publicationDate":"2023-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Nightmare as a trigger for stress cardiomyopathy\",\"authors\":\"Marc Arcens, Stephane Noble\",\"doi\":\"10.5603/cj.97167\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"A 67-year-old female was admitted to the emergency department with moderate dyspnea that escalated into oppressive chest pain after 4 hours. She had mild hypertension (153/107 mmHg) and sinus tachycardia (110 bpm). The electrocardiogram showed a left axis deviation without any signs of ischemia and a corrected QT interval of 471 ms (Fig. 1A). The biological markers showed a N-teminal-pro-B-type natriuretic peptide level of 1513 ng/L, peak troponins of 3502 ng/L, and peak creatine kinase of 306 U/L. The transthoracic echocardiogram (TTE) showed severe apical dysfunction with a left ventricular ejection fraction (LVEF) of 25% (Fig. 1B, Suppl. Video 1, Part 1 ). A left ventriculography (Fig. 1C, Suppl. Video 1, Part 2 ) and coronary angiography (Fig. 1D, E)\",\"PeriodicalId\":93923,\"journal\":{\"name\":\"Cardiology journal\",\"volume\":null,\"pages\":null},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2023-12-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Cardiology journal\",\"FirstCategoryId\":\"0\",\"ListUrlMain\":\"https://doi.org/10.5603/cj.97167\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Cardiology journal","FirstCategoryId":"0","ListUrlMain":"https://doi.org/10.5603/cj.97167","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
A 67-year-old female was admitted to the emergency department with moderate dyspnea that escalated into oppressive chest pain after 4 hours. She had mild hypertension (153/107 mmHg) and sinus tachycardia (110 bpm). The electrocardiogram showed a left axis deviation without any signs of ischemia and a corrected QT interval of 471 ms (Fig. 1A). The biological markers showed a N-teminal-pro-B-type natriuretic peptide level of 1513 ng/L, peak troponins of 3502 ng/L, and peak creatine kinase of 306 U/L. The transthoracic echocardiogram (TTE) showed severe apical dysfunction with a left ventricular ejection fraction (LVEF) of 25% (Fig. 1B, Suppl. Video 1, Part 1 ). A left ventriculography (Fig. 1C, Suppl. Video 1, Part 2 ) and coronary angiography (Fig. 1D, E)