硬脑膜动静脉瘘所致严重非搏动性耳鸣的自杀意念

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摘要

搏动性耳鸣往往以硬脑膜动静脉瘘为首发表现,因此需要高度的怀疑,以避免误诊和可能的致命后果。虽然搏动性耳鸣通常起源于血管畸形和非搏动性耳鸣,但搏动性和非搏动性耳鸣可由其他潜在病理引起。如果血管病变引起的非搏动性主诉不能被检查人员听到,也不能被临床或放射学检测到,则必然会被误诊为中枢性耳鸣。在此,我们报告一个罕见的硬脑膜动静脉瘘导致主观,非搏动性耳鸣。本病例患者为50岁男性,耳鸣2年,多次治疗无效,最终产生自杀意念。头部磁共振成像、头颈部磁共振血管造影和颞骨计算机断层扫描未发现任何急性病理。脑血管造影显示左乙状窦及横窦有硬脑膜动静脉畸形。采用经动脉Onyx18栓塞术治疗硬脑膜动静脉畸形。治疗后症状缓解50%,6个月再评估时症状缓解80%。硬脑膜动静脉瘘是一种罕见的血管畸形,如果不及时治疗,会有致命的并发症。这些血管畸形可能无法通过体检或听力评估单独检测到。在没有神经功能缺损和逆行小脑膜或皮质静脉引流的情况下,选择性血管造影栓塞似乎是一种有效和低风险的对症治疗方式。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Suicidal Ideation in a Severe Case of NonPulsatile Tinnitus Caused by a Dural Arteriovenous Fistula
Abstract Pulsatile tinnitus is often the initial presentation of a dural arteriovenous fistula, so a high index of suspicion is needed to avoid misdiagnoses and possible fatal consequences. Although pulsatile tinnitus commonly originates from a vascular malformation and non-pulsatile tinnitus from a neurodegenerative one, pulsatile and non-pulsatile presentations can result from other underlying pathologies. If a vascular pathology causes a non-pulsatile complaint that cannot be heard by the examiner or detected clinically or radiologically, it is bound to be misdiagnosed as central tinnitus. Herein, we report a rare presentation of a dural arteriovenous fistulas contributing to subjective, non-pulsatile tinnitus. The 50-year-old male patient in this case presented with 2 years of tinnitus, refractory to several treatment approaches that eventually lead to suicidal ideation. Magnetic resonance imaging of the head, magnetic resonance angiography of the head and neck, and computerized tomography scan of the temporal bone did not reveal any acute pathology. Cerebral angiogram revealed a dural arterio-venous malformation in the left sigmoid and transverse sinus. The patient was treated with transarterial Onyx18 embolization of the dural arterio-venous malformation. His symptoms resolved by 50% following treatment and by 80% at 6-month reevaluation. Dural arteriovenous fistulas are rare vascular malformations that, if left untreated, have fatal complications. These vascular malformations may not be detected by physical examination or audiometric evaluation alone. In the absence of neurologic deficits and retrograde leptomeningeal or cortical venous drainage, selective angiographic embolization appears to be an efficient and low-risk modality for symptomatic treatment.
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