阿育吠陀治疗杜氏肌萎缩症与提高生活质量的相关性-一份儿科病例报告

IF 0.2
Renu Rathi, Sakshi Shirbhate, Bharat Rathi, Sadhana Misar, Monali Kurhadkar, Prasad Yewle
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引用次数: 0

摘要

肌萎缩症是一种以进行性肌肉无力为临床特征的肌病。杜氏肌营养不良症(DMD)是儿童肌营养不良最常见的x连锁疾病,存在于儿童早期,其特征是受影响男孩的近端肌肉无力和小腿肥大。这个案例旨在让阿育吠陀儿科医生意识到为DMD制定方案。目的是在一定程度上提供运动功能的即时缓解,并提高生活质量。一个11岁的男孩表现出站立能力不足,肌肉无力,下肢运动困难,特别是在所有剧烈的身体活动中。他还出现力量、耐力下降和进行性虚弱,伴有高尔氏征,提示DMD。目前还没有确定的经济治疗方案来预防疾病的进展和死亡率。对家庭成员进行了咨询,并保证改善他们的生活质量。他开始进行多次panchakarma前治疗,交替服用Rukshan和Brihan,以安抚内部阿育吠陀药物,并在不同坐姿的职业治疗和物理治疗的支持下进行三个月。CPK -肌酐激酶为8335 U/L (NV= 55 ~ 170 U/L),处理后变为4741u /L。由于挛缩减少,他可以独自站立15分钟,身体活动自如。由于这种遗传疾病没有药物治疗,测角仪测量的运动范围增加。然而,通过采用多维的综合方法、家庭支持和意志力,生活质量可以在很大程度上得到提高。阿育吠陀在巨大的改善中起着至关重要的作用,因为panchkarma萨满程序、药物、瑜伽、物理治疗、牵引和终身定期咨询。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Relevance of Ayurveda Management in Duchenne Muscular Dystrophy to Augment the Quality of Life- A Pediatric Case Report
Muscular dystrophy is a myopathy that stakes clinical characteristics of progressive muscular feebleness. Duchenne muscular dystrophy (DMD) is the most common X-linked disorder of muscular dystrophy in children, present in early childhood, and characterized by proximal muscle weakness and calf hypertrophy in affected boys. This case aims to make Ayurveda pediatricians aware of planning a protocol for DMD. The objective is to provide instant relief in motor function to some extent and to augment the quality of life. An 11 years old boy was presented with an in ability to stand, gross muscle weakness, difficulty in movements of lower limbs, and particularly in all vigorous physical activities. He also had decreased strength, stamina, and progressive debility with positive Gower's sign, which suggested DMD. Confirmed economic treatment options are unavailable to prevent progressive illness and mortality. The family members were counselled and assured of improving their quality of life. He was started with multiple pre-panchakarma procedures of alternate Rukshan and Brihan to pacify internal Ayurveda medicines with the support of occupational therapy and physiotherapy in different sittings for three months. His CPK -creatinine kinase was 8335 U/L (NV= 55 to 170 U/L) which became4741U/L post-treatment. He could stand with support for 15 minutes on his own with ease in body movements due to a reduction in contractures & increase in the range of motions measured by the goniometer, as this genetic disease has no medicinal cure. However, by adopting a multi-dimensional integrative approach, family support, and willpower, the quality of life can be augmented to much extent. Ayurveda plays a crucial role in the drastic improvement because of panchkarma shaman procedures, medicines, Yog, physiotherapy, traction, and counselling at regular intervals for lifelong.
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