基因行为理论:从零到有形糖基化的行为对癌症预后和治疗策略的影响

S. Mohamed
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引用次数: 0

摘要

一些社区疾病易感性的增加给卫生规划人员和提供者带来了问题。先前的基因-行为理论概述了非编码卫星DNA的行为与疾病易感性之间的因果关系。然而,这一理论观点需要进一步思考,以了解更多的这种关系;它确实为进一步严格的研究提供了一个平台。在这篇综述中,我们从新的角度对糖基化进行了回顾,因此我们使用顺序推理来关注和追踪它与卫星DNA和癌症易感性的关系。我们的模型表明糖基化是由行为引起的卫星DNA改变的主要有形行动。我们的模型还表明糖基化受到遗传预期的影响。此外,糖基化模式可以作为社会科学、社区靶向方法和行为相关发病机制的早期预后工具的行为生物标志物。这些概念为行为免疫遗传学和行为流行病学领域开辟了新的途径。因此,需要更深入和改进的治疗策略,特别是对癌症。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Gene-behavior theory: Behavior from nil to tangible glycosylation-implications for cancer prognosis and treatment strategies
Increased disease susceptibility in some communities is problematic for health planners and providers. The previous gene-behavior theory outlined a causal relationship between behavior and disease susceptibility in non-coding satellite DNA. While this theoretical viewpoint requires further thought to know more about this relationship; it does provide a platform for further rigorous research. In this review, glycosylation was reviewed from a new perspective, thus we focused and tracked its association with satellite DNA and cancer susceptibility using sequential reasoning. Our model suggests glycosylation is a major tangible action of satellite DNA alterations caused by behaviors. Our model also suggests glycosylation is influenced by genetic anticipation. In addition, glycosylation patterns may function as behavioral biomarkers for the social sciences, community-targeted approaches, and early prognostic tools for behavioral-related pathogenesis. These notions open up new avenues for behavioral immunogenetics and behavioral epidemiology areas. Therefore, more in-depth and improved treatment strategies are required, especially for cancer.
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