atp酶对营养原的作用- mtor释放长脂肪酸链,在线粒体磷脂酶、合成酶和合成酶的作用下形成3种ror - α、β、γ亚型

A. Tantawi
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引用次数: 0

摘要

RORs同工型是脂质代谢和脂肪生物合成中非常活跃的生物分子,强烈依赖于并受OPA1线粒体基因及其活性线粒体酶的调节,其中每种线粒体酶(磷脂酶、合成酶和合成酶)负责其自身的ROR同工型{磷脂酶负责ROR- α合成,合成酶负责ROR[1] β合成,以及负责ror - γ合成的合成酶,作用并发挥长脂肪酸分子的作用,这些长脂肪酸分子是由atp酶和COX酶对脂质分子的作用产生的,脂质分子伴随并与吸收的营养分子(pro- lipop -nutrient -mTOR分子)相关,然后按照自己的途径进行脂肪和氨基酸的生物合成,进行主动抗[1]炎症的生物合成,然后在原始细胞增殖中按照自己的功能。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
ATPase Effects on pro-nutrients-mTOR release long fatty acids chains which under mitochondrial phospholipase, synthase & synthetase effects form three ROR-alpha, beta, gamma isoform
RORs isoforms are so active biological molecules in lipid metabolism and in fat biosynthesis, that strongly dependent on and regulated by OPA1 mitochondrial genes and its active mitochondrial enzymes where each of mitochondrial enzyme (phospholipase, synthase, and synthetase) is responsible for its own ROR isoform {phospholipase responsible for ROR-alpha synthesis, synthase responsible for ROR[1]beta synthesis, and synthetase responsible for ROR-gamma synthesis} for acting and functioning the long fatty acids molecules “which produced from the effects of ATPase and COX enzyme on lipid molecules which accompanied and associated with absorbed nutrient molecules (pro-lipo-nutrient -mTOR molecules) “, and then will follow its own pathway in fatty and amino acids biosynthesis, in active anti[1]inflammations biosynthesis, and then will follow its own functions in original cells proliferations.
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