Y. Yoon, H. Sim, J. Kim, D. Park, J. Sohn, B. Chun, M. J. Kim
{"title":"甲型肝炎感染合并急性肾损伤的临床特征及VP1区序列分析","authors":"Y. Yoon, H. Sim, J. Kim, D. Park, J. Sohn, B. Chun, M. J. Kim","doi":"10.5145/KJCM.2010.13.1.7","DOIUrl":null,"url":null,"abstract":"was independently associated with AKI occurrence in non-fulminant HAV infections (odds ratios=1.094, 95% confidence interval=1.011−1.183). HAV RNA was detected in 57 (86.4%) patients: 53 strains (93.0%) showed genotype IIIA and 4 strains pre- sented genotype IA. All HAV isolates from the AKI patients belonged to the genotype IIIA and shared the identical sequences with those from the non-AKI patients. Conclusion: This study indicates that higher level of C-reactive protein is associated with AKI occurrence in non-fulminant HAV infections. There were no spe- cific nucleotide or amino acid substitutions in the VP1 region associated with AKI occurrence. (Korean J Clin Microbiol 2010;13:7-13)","PeriodicalId":143093,"journal":{"name":"Korean Journal of Clinical Microbiology","volume":"18 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2010-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"4","resultStr":"{\"title\":\"Clinical Characterization of Hepatitis A Infection Complicated with Acute Kidney Injury and Sequence Analysis of the VP1 Region\",\"authors\":\"Y. Yoon, H. Sim, J. Kim, D. Park, J. Sohn, B. Chun, M. J. Kim\",\"doi\":\"10.5145/KJCM.2010.13.1.7\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"was independently associated with AKI occurrence in non-fulminant HAV infections (odds ratios=1.094, 95% confidence interval=1.011−1.183). HAV RNA was detected in 57 (86.4%) patients: 53 strains (93.0%) showed genotype IIIA and 4 strains pre- sented genotype IA. All HAV isolates from the AKI patients belonged to the genotype IIIA and shared the identical sequences with those from the non-AKI patients. Conclusion: This study indicates that higher level of C-reactive protein is associated with AKI occurrence in non-fulminant HAV infections. There were no spe- cific nucleotide or amino acid substitutions in the VP1 region associated with AKI occurrence. (Korean J Clin Microbiol 2010;13:7-13)\",\"PeriodicalId\":143093,\"journal\":{\"name\":\"Korean Journal of Clinical Microbiology\",\"volume\":\"18 1\",\"pages\":\"0\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2010-03-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"4\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Korean Journal of Clinical Microbiology\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.5145/KJCM.2010.13.1.7\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Korean Journal of Clinical Microbiology","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.5145/KJCM.2010.13.1.7","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Clinical Characterization of Hepatitis A Infection Complicated with Acute Kidney Injury and Sequence Analysis of the VP1 Region
was independently associated with AKI occurrence in non-fulminant HAV infections (odds ratios=1.094, 95% confidence interval=1.011−1.183). HAV RNA was detected in 57 (86.4%) patients: 53 strains (93.0%) showed genotype IIIA and 4 strains pre- sented genotype IA. All HAV isolates from the AKI patients belonged to the genotype IIIA and shared the identical sequences with those from the non-AKI patients. Conclusion: This study indicates that higher level of C-reactive protein is associated with AKI occurrence in non-fulminant HAV infections. There were no spe- cific nucleotide or amino acid substitutions in the VP1 region associated with AKI occurrence. (Korean J Clin Microbiol 2010;13:7-13)
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