{"title":"克鲁松综合征和眼睛","authors":"Anushree Gupta","doi":"10.4103/hjo.hjo_22_22","DOIUrl":null,"url":null,"abstract":"Crouzon syndrome is characterized by premature fusion of cranial sutures (craniosynostosis) which can impair proper brain and craniofacial development. Craniofacial abnormalities include frontal bossing, midface hypoplasia, parrot beak nose, and hypoplastic maxilla. The most common ophthalmic manifestations of Crouzon syndrome are proptosis, exotropia, exposure keratopathy, hypertelorism, and optic atrophy secondary to chronic papilledema. Here we report a case of Crouzon syndrome with ocular complications.","PeriodicalId":370883,"journal":{"name":"Himalayan Journal of Ophthalmology","volume":"420 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2023-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Crouzon syndrome and the eye\",\"authors\":\"Anushree Gupta\",\"doi\":\"10.4103/hjo.hjo_22_22\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Crouzon syndrome is characterized by premature fusion of cranial sutures (craniosynostosis) which can impair proper brain and craniofacial development. Craniofacial abnormalities include frontal bossing, midface hypoplasia, parrot beak nose, and hypoplastic maxilla. The most common ophthalmic manifestations of Crouzon syndrome are proptosis, exotropia, exposure keratopathy, hypertelorism, and optic atrophy secondary to chronic papilledema. Here we report a case of Crouzon syndrome with ocular complications.\",\"PeriodicalId\":370883,\"journal\":{\"name\":\"Himalayan Journal of Ophthalmology\",\"volume\":\"420 1\",\"pages\":\"0\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2023-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Himalayan Journal of Ophthalmology\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.4103/hjo.hjo_22_22\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Himalayan Journal of Ophthalmology","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.4103/hjo.hjo_22_22","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Crouzon syndrome is characterized by premature fusion of cranial sutures (craniosynostosis) which can impair proper brain and craniofacial development. Craniofacial abnormalities include frontal bossing, midface hypoplasia, parrot beak nose, and hypoplastic maxilla. The most common ophthalmic manifestations of Crouzon syndrome are proptosis, exotropia, exposure keratopathy, hypertelorism, and optic atrophy secondary to chronic papilledema. Here we report a case of Crouzon syndrome with ocular complications.
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