耳廓小叶瘢痕疙瘩去除术

Syarif Fr, Lestari S, Monica Pw, Syarif Sk
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引用次数: 0

摘要

瘢痕疙瘩是由伤口愈合异常引起的纤维增生性病变,其特征是胶原沉积过多,不能自发愈合。对于医生来说,治疗耳内瘢痕疙瘩是一种具有挑战性的疾病,尤其是那些大的。这种情况对患者有心理社会影响,尽管瘢痕疙瘩复发的可能性很高,但对外观的考虑往往是患者寻求治疗瘢痕疙瘩的主要原因。在各种瘢痕疙瘩治疗方案中,可以做的一种方法是手术切除治疗和减积技术。病例报告:报告1例左耳小叶瘢痕疙瘩,尺寸为2.5 cm × 3 cm,患者行耳洞穿刺后出现轻微瘙痒和审美障碍。瘢痕疙瘩在穿孔后6个月扩大。患者行局部麻醉Pehacain®(利多卡因2% +肾上腺素1:8万)减体积切除手术。在伤口关闭结束时,给予曲安奈德酮40毫克。拆线后一周给药,每两周重复一次,共4次。手术切除效果良好,患者满意。结论:考虑到病变面积大,并保证病变部位无瘢痕组织残留,应采用减积术切除。切除后立即注射皮质类固醇,减少成纤维细胞增殖、胶原蛋白和糖胺聚糖合成,抑制促炎介质,防止瘢痕疙瘩复发。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Keloid Excision with Debulking Technique on the Lobule Auricle Sinistra
Introduction: Keloids are fibroproliferative lesions caused by abnormal wound healing and are characterized by excessive collagen deposits that cannot heal spontaneously. Keloid in the ear is a challenging condition to be treated by doctors, especially the large ones. This condition has a psychosocial impact on the patient and a consideration for appearance is often the main reason for patients seeking to cure keloids, although there is a high possibility of keloid recurrence. Out of various keloid therapy options, one method that can be done is surgical excision therapy with debulking technique. Case report: A case of keloids in left ear lobule measuring 2.5 cm × 3 cm is reported after the patient does ear piercing with complaints of slight itch and aesthetical disturbance. Keloids enlarge 6 months after piercing. The case is treated by debulking excision surgery using local anesthesia Pehacain® (lidocaine 2% + adrenaline 1:80,000). At the end of the wound closure, 40 mg of triamcinolone acetone is given. It is regiven one week after the stitches are removed and repeatedly every 2 weeks for 4 times. Excision surgical results are good and the patient feels satisfied. Conclusion: Excision surgery with debulking technique is chosen because of the large size of the lesion and to ensure there is no residual keloid tissue at the site of predilection. Corticosteroid injection is given immediately after excision to decrease fibroblast proliferation, collagen and glycosaminoglycan synthesis and suppress proinflammatory mediators in order to prevent keloid recurrence.
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