J O Davis, G A Stephens, R H Freeman, J M Deforrest
{"title":"钠耗尽犬肾性高血压发展过程中心排血量的变化。","authors":"J O Davis, G A Stephens, R H Freeman, J M Deforrest","doi":"10.1042/cs055221s","DOIUrl":null,"url":null,"abstract":"<p><p>1. Chronic renovascular hypertension developed in uninephrectomized, sodium-depleted dogs in association with a decrease in cardiac output. 2. With sodium and volume repletion of these animals, cardiac output returned to normal but the high level of arterial pressure was unchanged; consequently, the peripheral arterioles dilated. 3. These observations provide evidence against the theory of whole-body autoregulation.</p>","PeriodicalId":10672,"journal":{"name":"Clinical science and molecular medicine. Supplement","volume":"4 ","pages":"221s-223s"},"PeriodicalIF":0.0000,"publicationDate":"1978-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1042/cs055221s","citationCount":"2","resultStr":"{\"title\":\"Changes in cardiac output during the development of renal hypertension in sodium-depleted dogs.\",\"authors\":\"J O Davis, G A Stephens, R H Freeman, J M Deforrest\",\"doi\":\"10.1042/cs055221s\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><p>1. Chronic renovascular hypertension developed in uninephrectomized, sodium-depleted dogs in association with a decrease in cardiac output. 2. With sodium and volume repletion of these animals, cardiac output returned to normal but the high level of arterial pressure was unchanged; consequently, the peripheral arterioles dilated. 3. These observations provide evidence against the theory of whole-body autoregulation.</p>\",\"PeriodicalId\":10672,\"journal\":{\"name\":\"Clinical science and molecular medicine. Supplement\",\"volume\":\"4 \",\"pages\":\"221s-223s\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1978-12-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://sci-hub-pdf.com/10.1042/cs055221s\",\"citationCount\":\"2\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Clinical science and molecular medicine. Supplement\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.1042/cs055221s\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Clinical science and molecular medicine. Supplement","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1042/cs055221s","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Changes in cardiac output during the development of renal hypertension in sodium-depleted dogs.
1. Chronic renovascular hypertension developed in uninephrectomized, sodium-depleted dogs in association with a decrease in cardiac output. 2. With sodium and volume repletion of these animals, cardiac output returned to normal but the high level of arterial pressure was unchanged; consequently, the peripheral arterioles dilated. 3. These observations provide evidence against the theory of whole-body autoregulation.
求助内容：
应助结果提醒方式：
京公网安备 11010802042870号
