突变更新:PLEC序列变异谱和相关的凝集菌病。

IF 4.3 3区 材料科学 Q1 ENGINEERING, ELECTRICAL & ELECTRONIC
ACS Applied Electronic Materials Pub Date : 2022-12-01 Epub Date: 2022-07-29 DOI:10.1002/humu.24434
Hassan Vahidnezhad, Leila Youssefian, Nailah Harvey, Ali Reza Tavasoli, Amir Hossein Saeidian, Soheila Sotoudeh, Aida Varghaei, Hamidreza Mahmoudi, Parvin Mansouri, Nikoo Mozafari, Omid Zargari, Sirous Zeinali, Jouni Uitto
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引用次数: 3

摘要

Plectin由PLEC编码,是许多细胞类型中表达的中间细丝的细胞骨架连接物。Plectin由三个决定其功能的主要结构域组成:n端结构域、Rod结构域和c端结构域。与功能方面相关的PLEC分子缺陷导致一组罕见的遗传性疾病,即胸腺病。这些多系统疾病包括常染色体显性型单纯大疱性表皮松解症(EBS- ogna)、肢带状肌营养不良(LGMD)、先天性皮肤发育不全(ACC)和常染色体隐性型EBS,后者可能与肌肉营养不良(EBS- md)、幽门闭锁(EBS- pa)和/或先天性肌无力综合征(EBS- mys)有关。在这项研究中,通过下一代测序对600多名伊朗大疱性表皮松解症患者进行基因分型,鉴定出15名患有致病PLEC变异的患者。该突变更新分析了我们的队列和文献中PLEC的临床谱,并证明了PLEC基因型和表型表现之间的关系。本研究将我们的7个新的PLEC变异和表型发现与先前发表的116个变异数据结合起来,提供了最完整的PLEC致病性变异和相关疾病的概述。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Mutation update: The spectra of PLEC sequence variants and related plectinopathies.

Plectin, encoded by PLEC, is a cytoskeletal linker of intermediate filaments expressed in many cell types. Plectin consists of three main domains that determine its functionality: the N-terminal domain, the Rod domain, and the C-terminal domain. Molecular defects of PLEC correlating with the functional aspects lead to a group of rare heritable disorders, plectinopathies. These multisystem disorders include an autosomal dominant form of epidermolysis bullosa simplex (EBS-Ogna), limb-girdle muscular dystrophy (LGMD), aplasia cutis congenita (ACC), and an autosomal recessive form of EBS, which may associate with muscular dystrophy (EBS-MD), pyloric atresia (EBS-PA), and/or congenital myasthenic syndrome (EBS-MyS). In this study, genotyping of over 600 Iranian patients with epidermolysis bullosa by next-generation sequencing identified 15 patients with disease-causing PLEC variants. This mutation update analyzes the clinical spectrum of PLEC in our cohort and in the literature and demonstrates the relationship between PLEC genotype and phenotypic manifestations. This study has integrated our seven novel PLEC variants and phenotypic findings with previously published data totaling 116 variants to provide the most complete overview of pathogenic PLEC variants and related disorders.

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来源期刊
CiteScore
7.20
自引率
4.30%
发文量
567
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