烟草烟雾引起的慢性阻塞性肺疾病(COPD)的动物模型和机制。

IF 6.4 2区 医学 Q1 ENVIRONMENTAL SCIENCES
Priya Upadhyay, Ching-Wen Wu, Alexa Pham, Amir A Zeki, Christopher M Royer, Urmila P Kodavanti, Minoru Takeuchi, Hasan Bayram, Kent E Pinkerton
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引用次数: 0

摘要

慢性阻塞性肺疾病(COPD)是全世界第三大死亡原因,其全球卫生负担正在增加。慢性阻塞性肺病的特征是肺气肿、粘液分泌过多和持续的肺部炎症,临床表现为慢性气流阻塞,患者出现呼吸困难、咳嗽和疲劳等症状。慢性支气管炎、肺气肿、哮喘和以高血压和动脉粥样硬化形式出现的心血管疾病等一系列病理在COPD患者中不同程度地共存。慢性阻塞性肺病的潜在原因主要包括烟草使用,但也可能与暴露于空气污染物、生物质燃烧以及与工作场所有关的烟雾和化学品有关。虽然没有单一的动物模型可以模拟人类COPD的所有特征,但各种已发表的模型共同帮助我们提高了对COPD发生和持续发生的疾病过程的理解。在这篇综述中,COPD的发病机制和相关危险因素在不同的哺乳动物模型中进行了研究。本综述中包括的每个动物模型都是由烟草烟雾(TS)暴露产生的。随着动物模型继续帮助确定COPD的病理生物学机制和可能的新型治疗干预措施,我们讨论了每种动物模型的优缺点。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Animal models and mechanisms of tobacco smoke-induced chronic obstructive pulmonary disease (COPD).

Chronic obstructive pulmonary disease (COPD) is the third leading cause of death worldwide, and its global health burden is increasing. COPD is characterized by emphysema, mucus hypersecretion, and persistent lung inflammation, and clinically by chronic airflow obstruction and symptoms of dyspnea, cough, and fatigue in patients. A cluster of pathologies including chronic bronchitis, emphysema, asthma, and cardiovascular disease in the form of hypertension and atherosclerosis variably coexist in COPD patients. Underlying causes for COPD include primarily tobacco use but may also be driven by exposure to air pollutants, biomass burning, and workplace related fumes and chemicals. While no single animal model might mimic all features of human COPD, a wide variety of published models have collectively helped to improve our understanding of disease processes involved in the genesis and persistence of COPD. In this review, the pathogenesis and associated risk factors of COPD are examined in different mammalian models of the disease. Each animal model included in this review is exclusively created by tobacco smoke (TS) exposure. As animal models continue to aid in defining the pathobiological mechanisms of and possible novel therapeutic interventions for COPD, the advantages and disadvantages of each animal model are discussed.

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来源期刊
CiteScore
13.80
自引率
6.90%
发文量
13
审稿时长
>24 weeks
期刊介绍: "Journal of Toxicology and Environmental Health: Part B - Critical Reviews" is an academic journal published by Taylor & Francis, focusing on the critical examination of research in the areas of environmental exposure and population health. With an ISSN identifier of 1093-7404, this journal has established itself as a significant source of scholarly content in the field of toxicology and environmental health. Since its inception, the journal has published over 424 articles that have garnered 35,097 citations, reflecting its impact and relevance in the scientific community. Known for its comprehensive reviews, the journal also goes by the names "Critical Reviews" and "Journal of Toxicology & Environmental Health, Part B, Critical Reviews." The journal's mission is to provide a platform for in-depth analysis and critical discussion of the latest findings in toxicology, environmental health, and related disciplines. By doing so, it contributes to the advancement of knowledge and understanding of the complex interactions between environmental factors and human health, aiding in the development of strategies to protect and improve public health.
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