{"title":"Study directions and development of cognitive theory of depression.","authors":"Krystyna Golonka, Elżbieta Piątek, Ryszard Stach","doi":"10.12740/PP/OnlineFirst/161676","DOIUrl":null,"url":null,"abstract":"<p><p>Aaron T. Beck's cognitive model of depression became the basis for the analysis of cognitive factors as significant correlates and predictors of depression. Psychological mechanisms of depression related to the depressive cognitive triad, the specificity of cognitive schemas and errors in cognitive processing determined the directions of further research on depression. The presented analysis of selected studies confirms and extends Beck's assumptions about the role of cognitive factors in the development and maintenance of depressive symptoms. The analysis of psychological, neuroimaging and biochemical studies provides a broader perspective of understanding both the etiology and mechanisms that sustain the symptoms of depression. The development of neuroimaging research has broadened the knowledge about the brain mechanisms of depression - the presented research combine the theoretical constructs of the cognitive concept of depression with the characteristics of the activity of brain structures and their functional connections. The following conclusions seem to be particularly important for clinical practice: 1) impairment of the cognitive inhibition function in depression reduces the control of negative automatic thoughts - strengthening working memory is important; 2) different brain mechanisms for processing rewards and punishments in people suffering from depression and in healthy people explain the formation of self-esteem - this knowledge can be an important element of psychoeducation in therapeutic work; 3) neuroimaging studies indicate neuronal correlates of cognitive distortions observed in depression - for further research on depression, it would be particularly important to monitor changes in brain activity in the course of its treatment.</p>","PeriodicalId":20863,"journal":{"name":"Psychiatria polska","volume":" ","pages":"669-680"},"PeriodicalIF":0.9000,"publicationDate":"2024-08-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Psychiatria polska","FirstCategoryId":"3","ListUrlMain":"https://doi.org/10.12740/PP/OnlineFirst/161676","RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q4","JCRName":"PSYCHIATRY","Score":null,"Total":0}
引用次数: 0
Abstract
Aaron T. Beck's cognitive model of depression became the basis for the analysis of cognitive factors as significant correlates and predictors of depression. Psychological mechanisms of depression related to the depressive cognitive triad, the specificity of cognitive schemas and errors in cognitive processing determined the directions of further research on depression. The presented analysis of selected studies confirms and extends Beck's assumptions about the role of cognitive factors in the development and maintenance of depressive symptoms. The analysis of psychological, neuroimaging and biochemical studies provides a broader perspective of understanding both the etiology and mechanisms that sustain the symptoms of depression. The development of neuroimaging research has broadened the knowledge about the brain mechanisms of depression - the presented research combine the theoretical constructs of the cognitive concept of depression with the characteristics of the activity of brain structures and their functional connections. The following conclusions seem to be particularly important for clinical practice: 1) impairment of the cognitive inhibition function in depression reduces the control of negative automatic thoughts - strengthening working memory is important; 2) different brain mechanisms for processing rewards and punishments in people suffering from depression and in healthy people explain the formation of self-esteem - this knowledge can be an important element of psychoeducation in therapeutic work; 3) neuroimaging studies indicate neuronal correlates of cognitive distortions observed in depression - for further research on depression, it would be particularly important to monitor changes in brain activity in the course of its treatment.
亚伦-贝克(Aaron T. Beck)的抑郁认知模型成为分析作为抑郁重要相关因素和预测因素的认知因素的基础。与抑郁认知三要素相关的抑郁心理机制、认知图式的特异性和认知处理过程中的错误决定了抑郁症进一步研究的方向。本文对部分研究的分析证实并扩展了贝克关于认知因素在抑郁症状的发展和维持中的作用的假设。对心理学、神经影像学和生化研究的分析为理解抑郁症状的病因和维持机制提供了更广阔的视角。神经影像学研究的发展拓宽了人们对抑郁症大脑机制的认识--这些研究将抑郁症认知概念的理论构建与大脑结构及其功能连接的活动特征相结合。以下结论似乎对临床实践尤为重要:1)抑郁症患者的认知抑制功能受损,降低了对消极自动思维的控制能力--加强工作记忆非常重要;2)抑郁症患者和健康人处理奖惩的大脑机制不同,解释了自尊的形成原因--这些知识可以成为治疗工作中心理教育的重要内容;3)神经影像学研究表明,抑郁症患者的认知扭曲与神经元相关--对抑郁症的进一步研究而言,监测治疗过程中大脑活动的变化尤为重要。