Hypomagnesemia, a Rare Cause of Reversible Ataxia.

IF 2.5 Q2 CLINICAL NEUROLOGY
Daniel López Domínguez, Juan Rodríguez Álvarez-Cienfuegos, Carla Herminia Vera Cáceres
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Abstract

Background: A 61-year-old male patient presented with cerebellar syndrome, which had progressively worsened for 10 days, followed by a tonic-clonic seizure.

Phenomenology shown: Blood analysis showed severe hypomagnesemia and a brain MRI showed T2 hyperintensity in the cerebellar hemispheres (Figure 1). Therefore, the final diagnosis was cerebellar syndrome and epileptic seizures secondary to severe hypomagnesemia.

Educational value: In cases of subacute onset of ataxia, the possibility of ataxia secondary to hypomagnesemia should be considered, as it can be diagnosed with a basic blood test and there are potentially life-threatening outcomes in the absence of treatment, with a reversible course following early supplementation.

Abstract Image

低镁血症,可逆性共济失调的罕见病因。
背景:一名61岁男性患者表现为小脑综合征,病情逐渐恶化10天,随后出现强直阵挛发作。现象学显示:血液分析显示严重低镁血症,脑MRI显示小脑半球T2高信号(图1)。因此,最终诊断为小脑综合征和继发于严重低镁血症的癫痫发作。教育价值:在亚急性发作的共济失调病例中,应考虑继发于低镁血症的共济失调的可能性,因为它可以通过基本的血液检查来诊断,并且在没有治疗的情况下存在潜在的危及生命的结果,在早期补充后具有可逆的过程。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
CiteScore
4.00
自引率
4.50%
发文量
31
审稿时长
6 weeks
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