Occupational stress and heart rate variability.

Q4 Medicine
M. Rauber, M. Bilban, R. Starc
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引用次数: 1

Abstract

Brief description of the article: This article considers heart rate variability as a measurable parameter of stress reaction and present recent studies that examined the impact of occupational stress on heart rate variability and thus autonomic nervous system. ABSTRACT Stress is a complex psychoneuroendocrinological and immune response of an individual to stressogenic factor. The most important contemporary stressogenic factors are mental, psycho-social and socio-economic stressors. This especially holds true for occupational stress. Many symptoms and signs of disease are associated with chronic occupational stress. Among those are cardiovascular diseases, metabolic and psychiatric disorders. This article describes different stressogenic factors that lead to occupational stress and two conceptual models of occupational stress (Karasek’s Demand/Control Model and Siegrist’s Effort-Reward Imbalance Model). Reaction to stress can be measured in various ways. This article summarizes the physiology of heart beat regulation and presents heart rate variability as a measurable parameter of stress reaction. Heart rate variability gives us an insight into autonomic modulation of the heart and functioning of the entire autonomic nervous system. This article presents the latest results of larger studies that examined the impact of occupational stress on heart rate variability and various mechanisms that lead to end-organ damage due to changes in autonomic nervous system as a result of work stress. Subjects exposed to chronic stress have significantly lower heart rate variability compared with subjects unaffected by stress. Researches have shown that reduced heart rate variability reliably predicts mortality among health adults, as well as morbidity and mortality in patients after acute myocardial infarction, in patients with chronic heart failure and mortality in patients with chronic kidney disease.
职业压力和心率变异性。
文章简介:本文认为心率变异性是应激反应的可测量参数,并介绍了最近的研究,研究了职业压力对心率变异性的影响,从而影响了自主神经系统。应激是个体对应激源因素的复杂心理神经内分泌和免疫反应。当代最重要的压力源因素是心理、心理社会和社会经济压力源。这尤其适用于职业压力。许多疾病的症状和体征都与慢性职业压力有关。其中包括心血管疾病、代谢和精神疾病。本文介绍了导致职业压力的不同压力源因素,以及职业压力的两种概念模型(Karasek的需求/控制模型和Siegrist的努力-回报不平衡模型)。对压力的反应可以用不同的方法来衡量。本文综述了心率调节的生理学,提出心率变异性是应激反应的可测量参数。心率变异性使我们对心脏的自主调节和整个自主神经系统的功能有了深入的了解。本文介绍了大型研究的最新结果,这些研究检查了职业压力对心率变异性的影响,以及由于工作压力导致自主神经系统变化而导致终末器官损伤的各种机制。与未受压力影响的受试者相比,暴露于慢性压力下的受试者心率变异性显著降低。研究表明,心率变异性的降低可以可靠地预测健康成年人的死亡率,以及急性心肌梗死后患者、慢性心力衰竭患者和慢性肾脏疾病患者的发病率和死亡率。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
CiteScore
0.30
自引率
0.00%
发文量
65
审稿时长
4-8 weeks
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