{"title":"Outpatient Thyroid surgery","authors":"B. Stack","doi":"10.4172/2167-7948.C1.003","DOIUrl":null,"url":null,"abstract":"H is a common endocrine disorder resulting from deficiency of Thyroid hormone. In the United States and other areas of adequate iodine intake, autoimmune Thyroid disease (Hashimoto disease) is the most common cause of hypoThyroidism; we can distinguish more types of Thyroiditis with primary hypoThyroidism, goiter or without, involved in poly-glandular autoimmune syndrome. Special normal Thyroid hormones values can be followed in pregnant women. Congenital hypoThyroidism, which affects 1 of every 4000 newborns, is due to congenital mal development of the Thyroid. This disorder is included in the newborn screening panel in many countries, and it is readily treatable once detected. Cretinism refers to severe hypoThyroidism in an infant or child. HypoThyroidism can be secondary—the Thyroid gland receives insufficient stimulation secretion of thyrotropin from the pituitary gland e.g. pituitaria adenoma, pituitary radiation, congenital, postpartum condition. In tertiary hypoThyroidism, inadequate secretion of thyrotropin-releasing hormone (TRH) from the hypothalamus leads to insufficient release of TSH, which in turn causes inadequate Thyroid stimulation. HypoThyroidism may also be drug-induced or otherwise iatrogenic. The patient’s presentation may vary from asymptomatic to myxedema coma with multisystem organ failure. Deficiency of the hormone has a wide range of effects. Classic signs and symptoms may not be present, especially in younger patients. Third-generation TSH assays are readily available and are generally the most sensitive screening tool for primary hypoThyroidism. The generally accepted reference range for normal serum TSH is 0.404.2 m IU/L. For hypoThyroidism, Thyroid hormone is administered to supplement or replace endogenous production. In general, hypoThyroidism can be adequately treated with a constant daily dose of levo thyroxine (LT4).","PeriodicalId":91565,"journal":{"name":"Journal of thyroid disorders & therapy","volume":"20 1","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2016-03-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"4","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of thyroid disorders & therapy","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.4172/2167-7948.C1.003","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 4
Abstract
H is a common endocrine disorder resulting from deficiency of Thyroid hormone. In the United States and other areas of adequate iodine intake, autoimmune Thyroid disease (Hashimoto disease) is the most common cause of hypoThyroidism; we can distinguish more types of Thyroiditis with primary hypoThyroidism, goiter or without, involved in poly-glandular autoimmune syndrome. Special normal Thyroid hormones values can be followed in pregnant women. Congenital hypoThyroidism, which affects 1 of every 4000 newborns, is due to congenital mal development of the Thyroid. This disorder is included in the newborn screening panel in many countries, and it is readily treatable once detected. Cretinism refers to severe hypoThyroidism in an infant or child. HypoThyroidism can be secondary—the Thyroid gland receives insufficient stimulation secretion of thyrotropin from the pituitary gland e.g. pituitaria adenoma, pituitary radiation, congenital, postpartum condition. In tertiary hypoThyroidism, inadequate secretion of thyrotropin-releasing hormone (TRH) from the hypothalamus leads to insufficient release of TSH, which in turn causes inadequate Thyroid stimulation. HypoThyroidism may also be drug-induced or otherwise iatrogenic. The patient’s presentation may vary from asymptomatic to myxedema coma with multisystem organ failure. Deficiency of the hormone has a wide range of effects. Classic signs and symptoms may not be present, especially in younger patients. Third-generation TSH assays are readily available and are generally the most sensitive screening tool for primary hypoThyroidism. The generally accepted reference range for normal serum TSH is 0.404.2 m IU/L. For hypoThyroidism, Thyroid hormone is administered to supplement or replace endogenous production. In general, hypoThyroidism can be adequately treated with a constant daily dose of levo thyroxine (LT4).
H是一种常见的由甲状腺激素缺乏引起的内分泌紊乱。在美国等碘摄入充足的地区,自身免疫性甲状腺疾病(桥本病)是甲状腺功能减退的最常见原因;我们可以区分更多类型的甲状腺炎与原发性甲状腺功能减退,甲状腺肿或无,涉及多腺自身免疫综合征。孕妇可以遵循特殊的正常甲状腺激素值。先天性甲状腺功能减退症是由先天性甲状腺发育不良引起的,每4000名新生儿中就有1名患有这种疾病。在许多国家,这种疾病被列入新生儿筛查小组,一旦发现就很容易治疗。克汀症是指婴儿或儿童严重的甲状腺功能减退症。甲状腺功能减退可以是继发性的——甲状腺从垂体获得的促甲状腺素分泌刺激不足,如垂体腺瘤、垂体放射、先天性、产后疾病。在三期甲状腺功能减退症中,下丘脑促甲状腺素释放激素(TRH)分泌不足导致TSH释放不足,进而导致甲状腺刺激不足。甲状腺功能减退也可能是药物引起的或其他医源性的。患者的表现从无症状到黏液性水肿昏迷伴多系统器官衰竭不等。缺乏这种激素会产生一系列的影响。典型的体征和症状可能不存在,特别是在年轻患者中。第三代TSH测定是现成的,通常是最敏感的筛查工具,原发性甲状腺功能减退。一般接受的正常血清TSH参考范围为0.404.2 m IU/L。对于甲状腺功能减退症,给予甲状腺激素以补充或替代内源性生产。一般来说,甲状腺功能减退症可以通过每日恒定剂量的左旋甲状腺素(LT4)得到充分治疗。