How excess dietary saturated fats induce insulin resistance

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引用次数: 2

Abstract

Excess dietary saturated fatty acids can increase the risk of and progression of type 2 diabetes. We will explore the mechanisms by which excess saturated fatty acids can reduce insulin sensitivity, suppress insulin production of beta cells through glucolipotoxicity, raise blood glucose, and lessen energy production in cells. Higher dietary saturated fatty acids, especially palmitic acid, can reduce the number of insulin receptors to approximately one-half of their normal number. This contributes to hyperinsulinemia, elevated blood glucose, and reduced mitochondrial energy production. Higher dietary saturated fatty acids also interfere with the signaling between the insulin receptor and the glucose transporter. This reduces the amount of glucose that can enter the cell and increases the risk of elevated blood glucose. Excess dietary saturated fatty acids have been found to suppress insulin production of beta cells and also to stimulate apoptosis of beta cells. Higher dietary saturated fatty acids can reduce the ability of the cells to produce glycogen from glucose, thus lowering energy storage. Finally, higher dietary saturated fatty acids can reduce mitochondrial energy production. Conclusion: Reducing dietary saturated fatty acids may help clear blood of excess glucose in type 2 diabetes.
过量饱和脂肪如何诱发胰岛素抵抗
饮食中过量的饱和脂肪酸会增加2型糖尿病的风险和进展。我们将探索过量饱和脂肪酸降低胰岛素敏感性、通过糖脂毒性抑制β细胞产生胰岛素、升高血糖和减少细胞能量产生的机制。较高的膳食饱和脂肪酸,尤其是棕榈酸,可以将胰岛素受体的数量减少到正常数量的一半左右。这会导致高胰岛素血症,血糖升高,线粒体能量产生减少。较高的膳食饱和脂肪酸也会干扰胰岛素受体和葡萄糖转运蛋白之间的信号传导。这减少了可以进入细胞的葡萄糖量,并增加了血糖升高的风险。已经发现过量的膳食饱和脂肪酸抑制β细胞的胰岛素产生,也刺激β细胞的凋亡。较高的膳食饱和脂肪酸会降低细胞从葡萄糖中产生糖原的能力,从而降低能量储存。最后,较高的膳食饱和脂肪酸可以减少线粒体能量的产生。结论:减少膳食饱和脂肪酸有助于清除2型糖尿病患者血液中的过量葡萄糖。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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