The Dentistry Conference Houston

Abstract

Most early visual diagnostics of cranial nerves pathologies in the natural history of oral cavity and craniofacial disorders (demyelinating neuropathy, infections, and neuronal gliomatosis). Rapid restoration of immunity using the molecular method of biocybernetic clavitherapy by dr. Ferdynand Barbasiewicz. Clinical diagnostics of neuropathy Chronic infections with edema lead to activation of destructive psychosomatic mechanisms whereas painkillers administered over several days worsen the restricted circulation of blood within the infected venules. If the infection and swelling area involves the second or the third branch of the trigeminal nerve, enzyme metabolic function may be impaired at sub molecular level due to insufficient blood supply. This leads to the failure of aggregation of peripheral blood interferons acting as biocybernetic mediators responsible for subsequent aggregation of lipids, cytokines, endorphins, and other molecular complements making up the myelin sheath into the oligodendrocyte loss zone. As the result, the myelin sheath insulation of the trigeminal nerve fiber becomes deteriorated, leading to reduced nerve potentials in the decay latency period ,responsible for regulation of the enzyme metabolic function or the function of the pyramidal tract fiber dynamizing the skeletal muscles of the oral cavity and splachnocranium. Reduced blood supply is also observed within the sclerotome and periosteum of local dentition. Moreover, infections involving edemas disrupt the blood supply to the dermatome, connective tissue, neurotome, and myotome. Sentinel lymph nodes are also affected by inflammation. What happens when the infection involves the second or the third branch of the trigeminal nerve, with decay latency potentials being markedly reduced or even zeroed in EMG examinations? In such cases, pharmacotherapeutics listed above not only fail to treat, but instead exacerbate the infection as the result of e.g. venular ischemia being worsened. This may lead to accelerated, progressive infection-related pain reaction and pathological ischemic edema being expanded beyond the dentition level to effect sclerotomal and periosteal atrophization. After several (10+) days, hyperalgesicalgostic spots, slightly pink in color, sized 1 to 7 mm, may develop following the infection crisis above the second or third trigeminal nerve fibers. This finding provides a diagnostic information on the loss of the first myelin sheath layer with partial discharge of body’s nerve potentials with reduced decay latency levels. Progression of this skin symptom during persistent infection with continuous ischemia and edema will be associated with the loss of oligodendrocytes within the second, i.e. the last myelin sheath. This will lead to hypoesthetic, algodystrophic spot developing above the neuropathic site, with body’s own nerve potentials being discharged into organic molecules while decay latency may reach the zero value in EMG examinations. Notably, neuropathy of nerve fibers which control the enzyme metabolic function of cell division at the sclerotome and periosteal level results in metabolic atrophism of dentition beyond the local level, resulting in teeth crumbling like moist brick below the freezing temperature. In this condition, extraction remains the only option. Management and Clinical Description of the Molecular Method of Biocybernetic Clavitherapy in Dentistry In case of an early-stage infection, e.g. that of a mouth ulcer in the vicinity of gums, take a sharp toothpick and press (strongly yet non-invasively) several times onto acupuncture site GRT-27 located on the main posterior regulating meridian, along thee median line below the turbinate bone and just above the red lip border. Each time, hold the pick for several seconds with one second intervals. Archives in Biomedical Engineering & Biotechnology Volume 5-Issue 2 Citation: Ferdynand Barbasiewicz, Monika Michalska. The Dentistry Conference Houston. 5(2): 2021. ABEB.MS.ID.000607. DOI: 10.33552/ABEB.2021.05.000607. Page 2 of 4 Repeat 3-5 times a day. The procedure facilitates elimination of the mouth ulcer using patient’s own selective antibodies. In the case of physical trauma to the gum and the periosteum involving slight of the tooth, Use two sharp toothpicks to immediately perform dermovisceral compression at the dermatome and connective tissue level above the periodontal lesion site; at the same time, use two toothpick held in the other hand to compress the extrameridian point PaM (see Atlas, p. 94) responsible for elimination of paradontosis). Hold for 3-5 seconds, repeat several times a day over 3-5 days. Initially, this will cause some pain; however, it would resolve within several days along with the infection and the swelling. For several days, patients should try to chew food using the other side of their mouth. The proposed stimulation method will result in very good blood supply for successful, drug-free elimination of post-traumatic symptoms with initial edema. Similar symptoms facilitating the clinical diagnosis of neuropathy consist in the presence of demyelination plaque being preset within the brain, the motor neurons, and the peripheral nervous system. Notably, stress, frustration, and prolonged irritation disturb the function of thyroid and parathyroid glands; when disturbed, the glands shrink and secrete lower quantities of calcium and phosphorus into peripheral blood. – Stress-induced reduction in calcium and phosphorus ions being released from the parathyroids into peripheral blood is maintained is the main cause of reduced dietary intake of calcium and phosphorus upon digestion in the jejunum and ileum. This is the direct cause of reduced bone strength in some patients. Dietary supplements are ineffective in these cases. Clinical elimination of neuropathy Unpigmented, slightly pink-colored, algostic spot sized 1 to 3-7 mm observed within the maxillofacial area in the vicinity of the second or third branch of the trigeminal nerve or at other skin locations, either on the left or on the right, showing signs of hyperalgesia upon being impinged with a sharp toothpick is a manifestation of early-stage demyelinating neuropathy. It is a sign of the loss of the first myelin sheath layer, e.g. that of the second or the third branch of the trigeminal nerve, with partially reduced potentials being discharged into the organic molecules. In such cases, two sharp toothpicks should be use to compress the spots so as to stimulate increased blood supply with aggregation of interferons as biocybernetic mediators which, upon further stimulation, will trigger neurotransmitter-mediated accumulation of lipids, cytokines endorphins, and other hitherto unknown molecular endogenous factors so that the loss of oligodendrocytes forming the myelin sheath is reversed within several days. Following restoration of the immunity of the nerve fiber, own nerve potentials which regulate the enzyme metabolic function at submolecular level are restored to effective decay latency levels. Similar healing will also be observed within sclerotomal and periosteal cells in a relatively young person. On the other hand, white algo dystrophic plaque, hypoalgesic upon stimulation using two toothpicks or slight compression, develops as the result of ignorance and negligence on facial skin or in other locations, spots should be stimulated using two toothpicks until piercing pain, lasting a fraction of a second, is experienced by the patient to evidence forced blood supply that will result in neurotransmiter-mediated aggregation of interferons originating from nearby healthy cells. Other endogenous agents, including lipids, cytokines, endorphins, and hitherto unknown complements will be subsequently attracted to the hyperemic plaque. Following several days stimulation, oligodendrocyte cells comprising both layers of the myelin sheath will be restored and unimpaired neural transmission will resume for ongoing regeneration of sclerotomal and periodontal cells within permanent dentition (only in relatively young patients in good general health). Caution! Lack of sustained stimulation-related pain following some period of myelin sheath regeneration indicates a deficit of body’s own interferons in the peripheral blood. In such case, stimulation of biologically active points responsible for increased endogenous production of interferons should be attempted. To this end, the following points should be simulated (appropriate locations to be found in the Atlas of Clavitherapy): Pm-62, p. 70; Wż-34, p. 84; Pm-54, p. 66; auricle, U-25 and 28 (growth factor to be compressed simultaneously using 2 toothpicks with feedback stimulation of umbilical cord rim line simultaneously compressed using 2 toothpicks) 3 times for several days until the pain resolves (do not puncture the navel; thermal stimulation using no-flame moxibustion allowed). When no care is provided on time to infections involving edema, capillary veins become affected. At the early stage of infection, venular blood flow follows a reciprocal exchange pattern. In order to alleviate infection and edema surrounding a dental root, moderately, strong, non-invasive, perpendicular compression should be performed using the tip of a toothpick projected above the ischemically infected root. Depending on the extent of infection, the compression should last between 1 and 2-3 minutes until pain is resolved. The procedure may be performed by the patient themselves. Initially, pain will intensify. However, stimulation should loosen collagen fibers within the venules to restore the trace blood supply. Along with the inflowing blood, appropriate antibodies will be delivered for rapid alleviation of pain, edema, and infection and subsequent complete restoration of venular blood flow. As the result, the edema and infection can be resolved within about a quarter of an hour. Other stimulation spots include these at the inferior rim of the auricle, n