[Methemoglobinemia as a result of acute accidental “poppres” poisoning – a case report].

Przeglad lekarski Pub Date : 2016-01-01
Daria Schetz, Wojciech Waldman, Jacek Sein Anand
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Abstract

Recently we observe an increase in the number of intoxications due to xenobiotics that may induce acute methemoglobinemia. Our study showed a case of acute methemoglobinemia (MetHb 55..2%) in 25 – year old man. Clinical symptoms were caused by ingestion of “poppers” which was used by patient as “aphrodisiac”. Summary: Conclusions: 1. Oral intake of alkyl nitrites, including isobutyl nitrite, is burdened with the risk of life-threatening methemoglobinemia . 2. In the case of poisonings running with normal pO2, reduced level of oxygen saturation, clinical signs of cyanosis does not react to oxygen therapy, and “chocolate” tinge of blood methemoglobinemia should be suspected. 3. In every case of MetHb the use of antidote, which is methylene blue, should be considered. 4. The severity of cyanosis correlates with the level of methemoglobin.

[急性意外“poppres”中毒引起的高铁血红蛋白血症-一例报告]。
最近,我们观察到由于可能引起急性高铁血红蛋白血症的外源性药物中毒的数量增加。我们的研究显示急性高铁血红蛋白血症病例(MetHb 55.2%)在25岁的男性。临床症状是由患者误服“罂粟花”引起的,而“罂粟花”被患者当作“春药”使用。摘要:结论:1。口服烷基亚硝酸盐,包括异丁基亚硝酸盐,有发生危及生命的高铁血红蛋白血症的风险。2. 在pO2正常的中毒情况下,血氧饱和度降低,临床表现为紫绀,对氧治疗无反应,应怀疑血高铁血红蛋白呈“巧克力”色。3.在每一种甲氧麻黄碱病例中,都应考虑使用解毒剂,即亚甲基蓝。紫绀的严重程度与高铁血红蛋白的水平有关。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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