Mechanisms for preserved cerebrovascular autoregulation during hypertension in rats after sinoaortic denervation.

W T Talman, D N Dragon
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引用次数: 8

Abstract

1. Cerebral blood flow (CBF) and cerebrovascular resistance (CVR) autoregulate to higher levels of arterial pressure (AP) in rats after sinoaortic denervation (SAD) than in intact rats. 2. Potential mechanisms for this phenomenon were studied by monitoring CBF by laser flowmetry while increasing AP in rats after: (i) SAD; (ii) SAD plus bilateral removal of the superior cervical ganglia; (iii) SAD plus interruption of all renal vessels and nerves (renal isolation); (iv) SAD plus sympathectomy and renal isolation; and (v) intravenous treatment with the nitric oxide synthase inhibitor L-nitroarginine (LNA). 3. Compared to intact control rats autoregulation persisted to higher absolute levels of AP in SAD rats with isolation of the kidneys and resistance did not fall significantly below that in rats with SAD alone. 4. Effects of SAD on autoregulation were not altered by combining renal isolation with an interruption of sympathetics. 5. LNA did not affect the baroreflex but blocked a breakthrough of autoregulation even at a maximal mean AP (MAP) of 189 +/- 2 mmHg. 6. Breakthrough occurred in animals pretreated with LNA plus L-arginine but not in animals given LNA plus D-arginine. 7. These data are consistent with a role of the baroreflex in the expression of breakthrough and suggest that breakthrough may result from release of nitric oxide or a nitric oxide donor.

主动脉去神经后大鼠高血压期间保留的脑血管自身调节机制。
1. 主动脉去神经(SAD)后大鼠的脑血流量(CBF)和脑血管阻力(CVR)自动调节到高于正常大鼠的动脉压(AP)水平。2. 通过激光血流仪监测脑血流,同时增加大鼠的AP,研究了这一现象的潜在机制:(ii) SAD联合双侧切除颈上神经节;(三)SAD加上所有肾血管和神经的中断(肾隔离);(四)SAD加交感神经切除术和肾分离;静脉注射一氧化氮合酶抑制剂l -硝基精氨酸(LNA)。3.与完整的对照大鼠相比,分离肾脏的SAD大鼠的自身调节持续到更高的AP绝对水平,并且抵抗并未明显低于单独SAD大鼠。4. 联合肾分离和交感神经中断,SAD对自身调节的影响没有改变。5. LNA不影响气压反射,但即使在最大平均AP (MAP)为189 +/- 2 mmHg时,也阻碍了自动调节的突破。6. 用LNA + l -精氨酸预处理的动物出现了突破,但在给予LNA + d -精氨酸的动物中没有。7. 这些数据与压力反射在突破表达中的作用一致,表明突破可能是由一氧化氮或一氧化氮供体的释放引起的。
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