[Subacute subdural hematoma: report of 3 cases].

S Nomura, T Orita, T Tsurutani, A Izumihara
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Abstract

Three surgical cases of subacute subdural hematomas are reported considering the pathophysiology. All patients had head traumas and complained the worsenings of the headaches 7-14 days after the head traumas. Serial computed tomography scan (CT) revealed the expansions of the subdural hematomas with the change of the density from high to mixed. The surgical findings of the hematomas showed blood clots and liquid covered with thin membranes. Morinaga et al. suggested the etiology of the subacute subdural hematoma that the influx of cerebrospinal fluid (CSF) to the hematoma cavity through the teared arachnoid. Our macroscopic findings of the hematomas which had the CSFlike liquid supported this estimation. We suggest that subacute subdural hematoma should be differed from chronic subdural hematoma because it may have the proper mechanisms of the development. Mild acute subdural hematomas should be observed carefully because they may develop to be subacute subdural hematomas between 1 and 3 weeks after the onset.

亚急性硬膜下血肿3例报告
本文报道3例亚急性硬膜下血肿的手术治疗。所有患者均有头部外伤,并在外伤后7 ~ 14天主诉头痛加重。连续CT扫描显示硬膜下血肿扩张,密度由高变为混合。血肿的外科检查结果为覆盖着薄膜的血凝块和液体。Morinaga等人认为亚急性硬膜下血肿的病因是脑脊液(CSF)通过撕裂的蛛网膜流入血肿腔。我们对含有csf样液体的血肿的宏观观察结果支持了这一估计。我们认为亚急性硬膜下血肿应与慢性硬膜下血肿区分开来,因为亚急性硬膜下血肿可能有其特定的发展机制。轻度急性硬膜下血肿应仔细观察,因为它们可能在发病后1至3周发展为亚急性硬膜下血肿。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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