Stress-induced reduction of gastric acid in the rat is not associated with increased tissue somatostatin.

Acta biologica et medica Germanica Pub Date : 1982-01-01
W Engelhardt, P O Schwille, U Wolf
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Abstract

In zero, mildly and severely stressed rats, gastric acid secretion, aortal and portal venous gastrin, venous glucagon and somatostatin in gastric, duodenal mucosa and in pancreas were examined. Serum gastrin and gastric acid secretion are reduced markedly by both kinds of stress, whereas plasma glucagon rises steadily with stress. As somatostatin in the tissues of stressed rats is not different from unstressed controls, gastrin and gastric acid reduction may not be attributed to an endocrine or paracrine action of somatostatin.

应激诱导的大鼠胃酸减少与组织生长抑素增加无关。
零、轻度和重度应激大鼠胃、十二指肠黏膜和胰腺胃酸分泌、主动脉和门静脉胃泌素、静脉胰高血糖素和生长抑素的变化。两种应激均使血清胃泌素和胃酸分泌明显减少,而血浆胰高血糖素则随应激而稳定升高。由于应激大鼠组织中的生长抑素与非应激对照并无不同,胃泌素和胃酸的减少可能不是由于生长抑素的内分泌或旁分泌作用。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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