Mechanisms of Slow Ventricular Tachycardias in Acute Myocardial Infarction

Agustin Castellanos Jr. M.D. , Louis Lemberg M.D., F.C.C.P. , Azucena G. Arcebal M.D.
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引用次数: 39

Abstract

Unifocal ventricular rhythms with rates between 60 and 100 per minute were classified as slow ventricular tachycardias when occurring in patients with acute myocardial infarction. Analysis of selected electrocardiograms showed that the paroxysms were usually automatic, nonparasystolic and intermittent However, in one case, the arrhythmia resulted from the exit block of a faster, parasystolic, ventricular tachycardia. The first ectopic beat in each series appeared either as an escape or as a late extrasystole. Variations in rate attributed to a disturbance of automaticity and/or conductivity were frequently observed. The erratic and unpredictable behavior of this arrhythmia probably reflected the unstable condition of the injured cells during the early stages of myocardial infarction.

急性心肌梗死慢性室性心动过速的机制
急性心肌梗死患者发生的单灶性室性心动过速在每分钟60 - 100次之间时被归类为慢性室性心动过速。对所选心电图的分析表明,发作通常是自动的、非副收缩期的和间歇性的。然而,在一个病例中,心律失常是由于更快的、副收缩期的室性心动过速的出口阻断引起的。每个系列的第一次异位搏动要么表现为逃逸,要么表现为晚期的早搏。由于自动性和/或导电性的干扰,经常观察到速率的变化。这种心律失常的不稳定和不可预测的行为可能反映了心肌梗死早期损伤细胞的不稳定状态。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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