Restoring Cerebral Venous Outflow: Right Internal Jugular to Subclavian Vein Bypass After Ligation in Thoracic Outlet Syndrome.

IF 0.7
Dushan Miladinovic, Luca Borruso, Eshan Affan, Steven Dubenec
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Abstract

BackgroundIntracranial venous hypertension (IVH) is an underrecognised cause of persistent craniofacial symptoms, often presenting with nonspecific features such as pressure-type headaches, pulsatile tinnitus, and visual disturbances. This case illustrates a complex diagnostic dilemma in which symptoms classically attributed to Eagle Syndrome masked another vascular cause of intracranial venous hypertension. The patient provided informed consent for treatment and for the use of his clinical information in this case report.Case SummaryA 59-year-old woman presented with refractory craniofacial symptoms initially attributed to bilateral Eagle Syndrome. Despite bilateral stylodectomy, internal jugular vein (IJV) decompression, C1 transverse process resection, and right occipital sinus stenting, her symptoms persisted. During right-sided intervention for Eagle Syndrome, the right IJV was found to have been previously ligated at the clavicle during venous thoracic outlet syndrome surgery. Venous manometry demonstrated elevated pressures within the right IJV (32 mmHg) and torcula (29 mmHg), supporting IVH secondary to impaired venous outflow. Definitive treatment consisted of a right IJV-subclavian vein (SCV) bypass using a triple-panel great saphenous vein (GSV) graft to achieve an adequate conduit diameter. The postoperative course was complicated by early graft thrombosis and neck hematoma, requiring urgent hybrid open and endovascular reintervention. This included suction thrombectomy, angioplasty, sequential stenting of the bypass using bare-metal, covered, and dedicated venous stents, and open repair of a graft tear with bovine pericardial patch angioplasty. Following reintervention, the bypass remained patent on follow-up imaging, with complete resolution of headaches, pulsatile tinnitus, and visual symptoms.ConclusionThis case highlights the importance of comprehensive venous evaluation in patients with persistent craniofacial symptoms, particularly following thoracic outlet interventions. It also demonstrates the feasibility of triple-panel GSV grafting for central venous reconstruction and the value of a hybrid surgical-endovascular approach in managing complex venous complications.

恢复脑静脉流出:胸廓出口综合征结扎术后右颈内至锁骨下静脉分流术。
背景颅静脉高压(IVH)是一种未被充分认识的持续性颅面症状的原因,通常表现为非特异性特征,如压力型头痛、搏动性耳鸣和视觉障碍。这个病例说明了一个复杂的诊断困境,其中典型的鹰综合征症状掩盖了颅内静脉高压的另一个血管原因。患者在本病例报告中提供了对治疗和使用其临床信息的知情同意。病例总结一名59岁女性,最初因双侧鹰综合征出现难治性颅面症状。尽管双侧茎突切除、颈内静脉(IJV)减压、C1横突切除和右侧枕窦支架置入术,她的症状仍然存在。在鹰综合征的右侧介入治疗中,在静脉胸廓出口综合征手术中发现右侧IJV先前在锁骨处结扎过。静脉测压显示右侧IJV内(32 mmHg)和环内(29 mmHg)血压升高,支持继发于静脉流出受损的IVH。最终治疗包括右ijv -锁骨下静脉(SCV)旁路,使用三板大隐静脉(GSV)移植物获得足够的导管直径。术后因早期移植物血栓形成和颈部血肿而复杂化,需要紧急混合开放和血管内再干预。这包括抽吸取栓、血管成形术、使用裸金属、覆盖和专用静脉支架进行连续支架术,以及使用牛心包补片血管成形术开放式修复移植物撕裂。再次介入手术后,旁路手术在随访影像上保持通畅,头痛、脉动性耳鸣和视觉症状完全消失。结论本病例强调了对持续性颅面症状患者进行全面静脉评估的重要性,特别是在胸廓出口介入治疗后。这也证明了三面板GSV移植用于中心静脉重建的可行性,以及手术-血管内混合入路在处理复杂静脉并发症中的价值。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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