[Expression of Concern] RIP kinase‑mediated ROS production triggers XAF1 expression through activation of TAp73 in casticin‑treated bladder cancer cells.

IF 4.7 3区 医学 Q2 ONCOLOGY
Oncology reports Pub Date : 2026-11-01 Epub Date: 2026-09-18 DOI:10.3892/or.2026.9192
Yoon Hee Chung, Daejin Kim
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引用次数: 0

Abstract

Following the publication of this paper, it was drawn to the Editor's attention by a concerned reader that the JNK western blotting data shown in Fig. 2E on p. 1138 were strikingly similar to the JNK western blotting data shown in Fig. 4D on p. 1139, even though the experimental conditions reported for these figure parts were apparently different. The authors have been contacted by the Editorial Office to offer an explanation for the apparent re‑use of the same data in this paper, and the authors have replied to explain that these studies were performed several years ago, that they no longer have access to the original data, and moreover, that they are not in a position to repeat any of these experiments. Owing to the fact that the Editorial Office has been made aware of potential issues surrounding the scientific integrity of this paper, we are issuing an Expression of Concern to notify readers of this potential problem associated with the abovementioned data. [Oncology Reports 36: 1135‑1142, 2016; DOI: 10.3892/or.2016.4895].

在蓖麻素处理的膀胱癌细胞中,RIP激酶介导的ROS产生通过激活TAp73触发XAF1表达。
在本文发表后,一位关心的读者提请编辑注意,图2E(1138页)所示的JNK western blotting数据与图4D(1139页)所示的JNK western blotting数据惊人地相似,尽管这两个图部分报道的实验条件明显不同。编辑部联系了作者,要求他们对论文中明显重复使用相同数据做出解释,作者回复解释说,这些研究是几年前进行的,他们不再有权访问原始数据,而且,他们无法重复任何这些实验。由于编辑部已经意识到围绕本文科学完整性的潜在问题,我们发布了一份关注表达,以通知读者与上述数据相关的潜在问题。[肿瘤报告]36:1135‑1142,2016;DOI: 10.3892 / or.2016.4895]。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Oncology reports
Oncology reports 医学-肿瘤学
CiteScore
8.50
自引率
2.40%
发文量
187
审稿时长
3 months
期刊介绍: Oncology Reports is a monthly, peer-reviewed journal devoted to the publication of high quality original studies and reviews concerning a broad and comprehensive view of fundamental and applied research in oncology, focusing on carcinogenesis, metastasis and epidemiology.
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