Predictive value of bone metabolism disease examination and changes in proximal humeral bone density for intraoperative anchor loosening in patients.

IF 3.2 4区医学 Q3 ENDOCRINOLOGY & METABOLISM

Archives of Physiology and Biochemistry Pub Date : 2026-04-10 DOI:10.1080/13813455.2026.2657426

Wei Wei, Xiangyu Yao, Wanli Duan, Jiang Zhu

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引用次数: 0

Abstract

Background: With the intensification of population ageing, the incidence of bone metabolism-related diseases such as osteoporosis has significantly increased, becoming an important factor affecting the quality of life of the elderly.

Materials and methods: A total of 120 patients with rotator cuff tear (RCT) were selected as the research subjects. There were 65 male patients and 55 female patients. The aim of this study was to explore the predictive value of bone metabolism disease examinations and changes in proximal humeral bone density for intraoperative anchor loosening. The predictive value of proximal humeral bone density for intraoperative anchor loosening was analysed using the ROC curve.

Results: The increase in age and the decrease in bone density are closely related to risk of intraoperative anchor loosening.

Conclusion: The combination of bone metabolism disease examinations and proximal humeral bone density detection can provide important basis for clinical preoperative prediction of anchor loosening risk and formulation of individualised treatment plans.

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骨代谢疾病检查和肱骨近端骨密度变化对患者术中锚钉松动的预测价值。

背景：随着人口老龄化的加剧，骨质疏松等骨代谢相关疾病的发病率显著增加，成为影响老年人生活质量的重要因素。材料与方法：选取120例肩袖撕裂（RCT）患者作为研究对象。男性65例，女性55例。本研究的目的是探讨骨代谢疾病检查和肱骨近端骨密度变化对术中锚钉松动的预测价值。应用ROC曲线分析肱骨近端骨密度对术中锚钉松动的预测价值。结果：年龄的增加和骨密度的降低与术中锚钉松动的危险密切相关。结论：骨代谢疾病检查结合肱骨近端骨密度检测可为临床术前预测锚钉松动风险及制定个体化治疗方案提供重要依据。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Archives of Physiology and Biochemistry ENDOCRINOLOGY & METABOLISM-PHYSIOLOGY

CiteScore

6.90

自引率

3.30%

发文量

期刊介绍： Archives of Physiology and Biochemistry: The Journal of Metabolic Diseases is an international peer-reviewed journal which has been relaunched to meet the increasing demand for integrated publication on molecular, biochemical and cellular aspects of metabolic diseases, as well as clinical and therapeutic strategies for their treatment. It publishes full-length original articles, rapid papers, reviews and mini-reviews on selected topics. It is the overall goal of the journal to disseminate novel approaches to an improved understanding of major metabolic disorders. The scope encompasses all topics related to the molecular and cellular pathophysiology of metabolic diseases like obesity, type 2 diabetes and the metabolic syndrome, and their associated complications. Clinical studies are considered as an integral part of the Journal and should be related to one of the following topics: -Dysregulation of hormone receptors and signal transduction -Contribution of gene variants and gene regulatory processes -Impairment of intermediary metabolism at the cellular level -Secretion and metabolism of peptides and other factors that mediate cellular crosstalk -Therapeutic strategies for managing metabolic diseases Special issues dedicated to topics in the field will be published regularly.