RETRACTION: Long Noncoding RNA lnc-ABCA12-3 Promotes Cell Migration, Invasion, and Proliferation by Regulating Fibronectin 1 in Esophageal Squamous Cell Carcinoma

IF 2.8 3区生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY

Journal of cellular biochemistry Pub Date : 2026-02-08 DOI:10.1002/jcb.70081

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引用次数: 0

Abstract

RETRACTION: J. Ma, Y. Xiao, B. Tian, S. Chen, B. Zhang, J. Wu, Z. Wu, X. Li, J. Tang, D. Yang, Y. Zhou, H. Wang, M. Su, and W. Wang, “Long Noncoding RNA lnc-ABCA12-3 Promotes Cell Migration, Invasion, and Proliferation by Regulating Fibronectin 1 in Esophageal Squamous Cell Carcinoma,” Journal of Cellular Biochemistry 121, no. 2 (2020): 1374–1387, https://doi.org/10.1002/jcb.29373.

The above article, published online on 12 September 2019 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the authors; the journal Editor-in-Chief, Christian Behl; and Wiley Periodicals LLC. The retraction has been agreed due to concerns raised by third parties. Specifically, several instances of image duplication within and across Figures 3B and 7 C have been detected. The clarification and materials provided by the authors upon request did not address the concerns. Accordingly, the article is retracted as the editors have lost confidence in the integrity and accuracy of the whole body of data presented in the article and consider its conclusions invalid.

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摘要：长链非编码RNA lnc-ABCA12-3通过调节纤维连接蛋白1在食管鳞状细胞癌中促进细胞迁移、侵袭和增殖

引用本文：马建军，肖彦，田斌，陈淑娟，张斌，吴建军，吴志杰，李晓霞，唐建军，杨德东，周勇，王红红，苏明，王伟，“长链非编码RNA lnc-ABCA12-3通过调控纤维连接蛋白1促进食管癌细胞迁移、侵袭和增殖”，《细胞生物化学》，第21期，no。2 (2020): 1374-1387, https://doi.org/10.1002/jcb.29373.The上述文章于2019年9月12日在线发表于Wiley online Library (wileyonlinelibrary.com)，经作者同意撤回；杂志主编克里斯蒂安·贝尔；和Wiley期刊有限责任公司。由于第三方提出的担忧，已同意撤回。具体来说，在图3B和图7c内和图7c之间检测到几个图像重复的实例。提交人应要求所作的澄清和提供的材料没有解决这些问题。因此，由于编辑对文章中提供的整个数据的完整性和准确性失去信心，并认为其结论无效，因此文章被撤回。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Journal of cellular biochemistry 生物-生化与分子生物学

CiteScore

9.90

自引率

0.00%

发文量

164

审稿时长

1 months

期刊介绍： The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.