Development of the Sleeping Pills Receptivity and Involuntariness Scale-6 (SPRIS-6) to Assess Acceptance of Hypnotics Use.

IF 3.4 2区医学 Q2 CLINICAL NEUROLOGY

Nature and Science of Sleep Pub Date : 2025-09-26 eCollection Date: 2025-01-01 DOI:10.2147/NSS.S550404

Seockhoon Chung, Mohd Ashik Shahrier

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引用次数: 0

Abstract

Background: Assessing an individual's acceptance of sleep medications is valuable for clinicians, as it may be the starting point for discussing the treatment of insomnia, whether pharmacologically or nonpharmacologically. This study developed a rating scale for measuring psychological receptivity to hypnotics and validated it in a general population.

Methods: We conducted an anonymous survey in a general Korean population. First, we developed a rating scale for psychological receptivity to hypnotic use using exploratory factor analysis (EFA) in Sample I (N = 300), followed by confirmatory factor analysis (CFA) in Sample II (N = 300). Convergent validity was assessed using the Insomnia Severity Index (ISI), Glasgow Sleep Effort Scale (GSES), Adaptive Cognition and Behaviors about Sleep-6 (ACBS-6), and Acceptance and Action Questionnaire-II (AAQ-II).

Results: From the EFA, six survey items were selected from the collected items. The CFA among Sample II showed a good fit for the two-factor model (Factor 1: Involuntary nature of insomnia; Factor 2: Acceptance of sleep medication use) of our Sleeping Pills Receptivity and Involuntariness Scale-6 (SPRIS-6) (comparative fit index = 0.99, Tucker-Lewis index = 0.99, root-mean-square-error of approximation = 0.02, and standardized root-mean-square residual = 0.02). According to the multi-group CFA, the two-factor structure of the SPRIS-6 measures psychological receptivity to sleep medications in the same way, regardless of whether or not participants reported insomnia. Using McDonald's coefficient of 0.80, the two-factor structure of the SPRIS-6 demonstrated good internal consistency. Linear regression analysis showed that the ISI score was positively influenced by the SPRIS-6 (β = 0.16, p = 0.002), GSES (β = 0.49, p = 0.001), and AAQ-II (β = 0.18, p = 0.001), whereas it was inversely influenced by the ACBS-6 (β = -0.10, p = 0.037).

Conclusion: The SPRIS-6 is a reliable and valid rating scale that measures psychological receptivity to sleep medication use.

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安眠药接受性和非自愿性量表-6 （SPRIS-6）的编制以评估安眠药使用的接受性。

背景：评估个体对睡眠药物的接受程度对临床医生来说是有价值的，因为它可能是讨论失眠治疗的起点，无论是药理学还是非药理学。本研究开发了一种评估催眠心理接受度的量表，并在普通人群中进行了验证。方法：对韩国普通人群进行匿名调查。首先，我们在样本1 （N = 300）中使用探索性因子分析（EFA）开发了催眠使用的心理接受度评定量表，然后在样本2 （N = 300）中使用验证性因子分析（CFA）。采用失眠严重程度指数（ISI）、格拉斯哥睡眠努力量表（GSES）、睡眠-6适应性认知与行为量表（ACBS-6）和接受与行动问卷- ii （AAQ-II）评估收敛效度。结果：从EFA收集的项目中选出6个调查项目。样本II的CFA显示，我们的安眠药接受性和非自愿性量表-6 （SPRIS-6）的双因素模型（因子1：失眠的非自愿性质；因子2：接受睡眠药物使用）（比较拟合指数= 0.99，Tucker-Lewis指数= 0.99，近似均方根误差= 0.02，标准化均方根残差= 0.02）拟合良好。根据多组CFA， SPRIS-6的双因素结构以同样的方式测量睡眠药物的心理接受度，无论参与者是否报告失眠。麦当劳系数为0.80，表明SPRIS-6的双因子结构具有良好的内部一致性。线性回归分析显示，ISI评分受SPRIS-6 （β = 0.16, p = 0.002）、GSES （β = 0.49, p = 0.001）和AAQ-II （β = 0.18, p = 0.001）的正相关影响，而受ACBS-6 （β = -0.10, p = 0.037）的负相关影响。结论：SPRIS-6是一种可靠、有效的睡眠药物使用心理接受度评定量表。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Nature and Science of Sleep Neuroscience-Behavioral Neuroscience

CiteScore

5.70

自引率

5.90%

发文量

245

审稿时长

16 weeks

期刊介绍： Nature and Science of Sleep is an international, peer-reviewed, open access journal covering all aspects of sleep science and sleep medicine, including the neurophysiology and functions of sleep, the genetics of sleep, sleep and society, biological rhythms, dreaming, sleep disorders and therapy, and strategies to optimize healthy sleep. Specific topics covered in the journal include: The functions of sleep in humans and other animals Physiological and neurophysiological changes with sleep The genetics of sleep and sleep differences The neurotransmitters, receptors and pathways involved in controlling both sleep and wakefulness Behavioral and pharmacological interventions aimed at improving sleep, and improving wakefulness Sleep changes with development and with age Sleep and reproduction (e.g., changes across the menstrual cycle, with pregnancy and menopause) The science and nature of dreams Sleep disorders Impact of sleep and sleep disorders on health, daytime function and quality of life Sleep problems secondary to clinical disorders Interaction of society with sleep (e.g., consequences of shift work, occupational health, public health) The microbiome and sleep Chronotherapy Impact of circadian rhythms on sleep, physiology, cognition and health Mechanisms controlling circadian rhythms, centrally and peripherally Impact of circadian rhythm disruptions (including night shift work, jet lag and social jet lag) on sleep, physiology, cognition and health Behavioral and pharmacological interventions aimed at reducing adverse effects of circadian-related sleep disruption Assessment of technologies and biomarkers for measuring sleep and/or circadian rhythms Epigenetic markers of sleep or circadian disruption.