RETRACTION: Novel Oral Transforming Growth Factor-β Signaling Inhibitor Potently Inhibits Postsurgical Adhesion Band Formation

IF 4 2区生物学 Q2 CELL BIOLOGY

Journal of Cellular Physiology Pub Date : 2025-09-08 DOI:10.1002/jcp.70089

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引用次数: 0

Abstract

RETRACTION: A. Soleimani, F. Asgharzadeh, F. Rahmani, et al. “Novel Oral Transforming Growth Factor-β Signaling Inhibitor Potently Inhibits Postsurgical Adhesion Band Formation.” Journal of Cellular Physiology 235, no. 5 (2019): 1349-1357, https://doi.org/10.1002/jcp.29053.

The above article, published online on 17 July 2019 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Robert Heath; and Wiley Periodicals LLC. The retraction has been agreed due to concerns raised by the third party, which revealed multiple inconsistencies found in Figure 2(g) and Figure 4(a), and additional inconsistencies were found in Figure 1(d). The explanation and raw data provided by the authors could not sufficiently address these concerns. Overall, the editors have lost confidence in the integrity and reliability of the full body of data presented in the article and consider the conclusions of this manuscript substantially compromised. The authors disagree with the retraction.

查看原文本刊更多论文

缩回：新型口服转化生长因子-β信号抑制剂有效抑制术后粘连带形成

撤回：A. Soleimani， F. Asgharzadeh， F. Rahmani等。“新型口服转化生长因子-β信号抑制剂有效抑制术后粘连带形成。”细胞生理学杂志，第235期。5 (2019): 1349-1357, https://doi.org/10.1002/jcp.29053.The上述文章于2019年7月17日在线发表在Wiley在线图书馆（wileyonlinelibrary.com）上，经主编Robert Heath同意撤回；和Wiley期刊有限责任公司。由于第三方提出的担忧，已经同意撤回，图2(g)和图4(a)中发现了多个不一致之处，图1(d)中发现了其他不一致之处。作者提供的解释和原始数据不能充分解决这些问题。总的来说，编辑对文章中提供的全部数据的完整性和可靠性失去了信心，并认为这篇手稿的结论基本上受到了损害。作者不同意撤稿。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Journal of Cellular Physiology 生物-生理学

CiteScore

14.70

自引率

0.00%

发文量

256

审稿时长

1 months

期刊介绍： The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.