RETRACTION: A Shared Comparison of Diabetes Mellitus and Neurodegenerative Disorders

IF 2.8 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
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引用次数: 0

Abstract

RETRACTION: M. Morsi, A. Maher, O. Aboelmagd, D. Johar, and L. Bernstein, “A Shared Comparison of Diabetes Mellitus and Neurodegenerative Disorders,” Journal of Cellular Biochemistry 119, no. 2 (2018): 1249–1256, https://doi.org/10.1002/jcb.26261.

The above article, published online on 25 September 2017 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the authors; the journal Editor-in-Chief, Christian Behl; and Wiley Periodicals LLC. The retraction has been agreed upon due to an editorial error, which led to the duplicate publication of the same article. Since the article published later in time contains further revisions and additional co-authors, the authors wished to keep this version for the scholarly record [1]. The publisher and the authors regret this error.

Reference

[1] M. Morsi, F. Kobeissy, S. Magdeldin, A. Maher, O. Aboelmagd, D. Johar, and L. Bernstein, “A Shared Comparison of Diabetes Mellitus and Neurodegenerative Disorders,” Journal of Cellular Biochemistry 120, no. 9 (2019): 14318–14325, https://doi.org/10.1002/jcb.28094.

撤回:糖尿病和神经退行性疾病的共同比较
撤稿:M. Morsi, A. Maher, O. Aboelmagd, D. Johar和L. Bernstein,“糖尿病和神经退行性疾病的共同比较”,《细胞生物化学杂志》119期,第2期。2 (2018): 1249-1256, https://doi.org/10.1002/jcb.26261.The上述文章于2017年9月25日在线发表在Wiley online Library (wileyonlinelibrary.com)上,经作者同意撤回;杂志主编克里斯蒂安·贝尔;和Wiley期刊有限责任公司。由于编辑错误,导致同一篇文章重复发表,因此已同意撤回。由于后来发表的文章包含了进一步的修订和其他共同作者,作者希望保留这个版本作为学术记录[1]。出版商和作者对这个错误表示遗憾。参考文献[10]M. Morsi, F. Kobeissy, S. Magdeldin, A. Maher, O. Aboelmagd, D. Johar, L. Bernstein,“糖尿病和神经退行性疾病的共同比较”,细胞生物化学杂志,第120期。9 (2019): 14318-14325, https://doi.org/10.1002/jcb.28094。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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