Correction to “The Role of TREM-1 in Septic Myocardial Pyroptosis and Septic Cardiomyopathy In Vitro and In Vivo”
IF 4
2区 生物学
Q2 CELL BIOLOGY
引用次数: 0
Abstract
Chen Y, Mao L, Liu S, Huang S, Lin Q, Zeng M, Huang H, Sun X, Chen H, Huang J, Zhou G, Deng L. The role of TREM-1 in septic myocardial pyroptosis and septic cardiomyopathy in vitro and in vivo. J Cell Physiol. 2024 Dec;239(12):e31445. https://doi.org/10.1002/jcp.31445.
In Figure 5 of “The Role of TREM-1 in Septic Myocardial Pyroptosis and Septic Cardiomyopathy In Vitro and In Vivo,” the authors mistakenly used a scanning electron micrograph from the drug treatment group in Figure 5b (WT control). This has been replaced with the correct control image. The corrected Figure 5 is shown below.
The authors apologize for this error.
对“TREM-1在体外和体内脓毒性心肌焦亡和脓毒性心肌病中的作用”的修正
陈艳，毛丽，刘松，黄松，林强，曾敏，黄慧，孙旭，陈华，黄健，周刚，邓磊。TREM-1在脓毒性心肌焦亡和脓毒性心肌病中的作用。中国生物医学工程学报，2009;39(12):391 - 391。https://doi.org/10.1002/jcp.31445.In图5《The Role of TREM-1 in Vitro and in Vivo in Septic Myocardial pyptosis and脓毒性心肌病in Vitro and in Vivo》，作者错误地使用了图5b （WT对照）中药物治疗组的扫描电子显微图。这已被替换为正确的控制映像。更正后的图5如下所示。作者为这个错误道歉。
本文章由计算机程序翻译，如有差异，请以英文原文为准。
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来源期刊
CiteScore
14.70
自引率
0.00%
发文量
256
审稿时长
1 months
期刊介绍：
The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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