Misalignment of age clocks.

IF 5.4 2区 医学 Q1 GERIATRICS & GERONTOLOGY
Xiaoyue Mei, Hannaneh Kabir, Michael J Conboy, Irina M Conboy
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引用次数: 0

Abstract

Biological aging is a complex non-linear process, with markedly distinct starting and end points, yet the biomarkers of its progression remain elusive. A key assumption of most machine learning (ML) approaches for age clocks is that predictive biomedical features can be identified via mathematical transformations of data to favor a linear transition from start to end, even if they erase any natural biological pattern. It is given that expected correlations, e.g., time lived (age) and time left to live (mortality), would persist in such mathematically optimized models, biologically meaningful or not. Here, we further clarify the workings of the clocks, explain the trade-off between mathematical optimization and biological interpretability, and discuss a hallmark of aging, inflammaging, that age clocks struggle to detect. We expand on the negative consequences of incoherence in linear models where some DNA methylation (DNAm) features increase with aging and disease, while others correspondingly decrease, yet positive weights are assigned to both. We quantify the misalignment between major DNAm clocks and actual changes in DNAm, providing an interactive visualization of these errors for each model. We demonstrate that major conventional age clocks are both incoherent and skewed toward leukocyte fractions and that rectifying incoherence makes the model balanced and not skewed toward neutrophils and better detects inflammaging. We briefly outline non-linear ML age clocks and the advantages of identifying a natural trajectory of aging directly from the primary data.

年龄时钟错位。
生物衰老是一个复杂的非线性过程,具有明显不同的起点和终点,但其进展的生物标志物仍然难以捉摸。大多数用于年龄时钟的机器学习(ML)方法的一个关键假设是,预测性生物医学特征可以通过数据的数学转换来识别,从而有利于从头到尾的线性过渡,即使它们消除了任何自然的生物模式。假定预期的相关性,例如,寿命(年龄)和剩余寿命(死亡率),将在这种数学优化的模型中持续存在,无论是否具有生物学意义。在这里,我们进一步澄清了时钟的工作原理,解释了数学优化和生物可解释性之间的权衡,并讨论了衰老的标志,炎症,年龄时钟难以检测。我们扩展了线性模型中不一致性的负面影响,其中一些DNA甲基化(DNAm)特征随着年龄和疾病而增加,而另一些则相应减少,但两者都被赋予了正权重。我们量化了主要DNAm时钟与DNAm实际变化之间的偏差,为每个模型提供了这些误差的交互式可视化。我们证明了主要的传统年龄时钟既不连贯又偏向白细胞分数,并且纠正不连贯使模型平衡而不偏向中性粒细胞并更好地检测炎症。我们简要概述了非线性机器学习年龄时钟,以及直接从原始数据中识别自然衰老轨迹的优势。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
GeroScience
GeroScience Medicine-Complementary and Alternative Medicine
CiteScore
10.50
自引率
5.40%
发文量
182
期刊介绍: GeroScience is a bi-monthly, international, peer-reviewed journal that publishes articles related to research in the biology of aging and research on biomedical applications that impact aging. The scope of articles to be considered include evolutionary biology, biophysics, genetics, genomics, proteomics, molecular biology, cell biology, biochemistry, endocrinology, immunology, physiology, pharmacology, neuroscience, and psychology.
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