{"title":"Consumptıon of ultra-processed foods can accelerate age-related appearance of sarcopenıa.","authors":"Hatice Parlak Baskurt, Hulya Yardımcı","doi":"10.1007/s10522-025-10253-8","DOIUrl":null,"url":null,"abstract":"<p><p>Sarcopenia, the loss of muscle mass and function, commonly affects older adults and reduces their quality of life. Frequent consumption of ultra-processed foods, which contain mostly additives and very few natural foods, increases the risk of sarcopenia or accelerates its onset. A diet rich in ultra-processed foods may lead to inadequate nutrition and lower intake of many nutrients, such as protein, dietary fiber, vitamins A, C, E, zinc, selenium, magnesium, and iron. However, obesity may occur because of increased energy and saturated fat intake. Both conditions contribute to the risk of muscle mass loss. A diet poor in antioxidants may increase the risk of sarcopenia by increasing inflammation. The fact that consumption of ultra-processed food contributes to the risk of frailty in older adults may lead to increased physical weakness and falls. Therefore, minimizing the consumption of ultra-processed foods is important to reduce the risk of sarcopenia. Understanding the contribution of the nutrients in this group to the risk of sarcopenia will allow for more accurate nutritional recommendations for old age. In our world, where the elderly population is increasing, it is important to conduct studies that include healthy nutrition to make this process healthier and more prosperous.</p>","PeriodicalId":8909,"journal":{"name":"Biogerontology","volume":"26 3","pages":"112"},"PeriodicalIF":4.4000,"publicationDate":"2025-05-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12085392/pdf/","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Biogerontology","FirstCategoryId":"3","ListUrlMain":"https://doi.org/10.1007/s10522-025-10253-8","RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"GERIATRICS & GERONTOLOGY","Score":null,"Total":0}
引用次数: 0
Abstract
Sarcopenia, the loss of muscle mass and function, commonly affects older adults and reduces their quality of life. Frequent consumption of ultra-processed foods, which contain mostly additives and very few natural foods, increases the risk of sarcopenia or accelerates its onset. A diet rich in ultra-processed foods may lead to inadequate nutrition and lower intake of many nutrients, such as protein, dietary fiber, vitamins A, C, E, zinc, selenium, magnesium, and iron. However, obesity may occur because of increased energy and saturated fat intake. Both conditions contribute to the risk of muscle mass loss. A diet poor in antioxidants may increase the risk of sarcopenia by increasing inflammation. The fact that consumption of ultra-processed food contributes to the risk of frailty in older adults may lead to increased physical weakness and falls. Therefore, minimizing the consumption of ultra-processed foods is important to reduce the risk of sarcopenia. Understanding the contribution of the nutrients in this group to the risk of sarcopenia will allow for more accurate nutritional recommendations for old age. In our world, where the elderly population is increasing, it is important to conduct studies that include healthy nutrition to make this process healthier and more prosperous.
期刊介绍:
The journal Biogerontology offers a platform for research which aims primarily at achieving healthy old age accompanied by improved longevity. The focus is on efforts to understand, prevent, cure or minimize age-related impairments.
Biogerontology provides a peer-reviewed forum for publishing original research data, new ideas and discussions on modulating the aging process by physical, chemical and biological means, including transgenic and knockout organisms; cell culture systems to develop new approaches and health care products for maintaining or recovering the lost biochemical functions; immunology, autoimmunity and infection in aging; vertebrates, invertebrates, micro-organisms and plants for experimental studies on genetic determinants of aging and longevity; biodemography and theoretical models linking aging and survival kinetics.