Correction to “Ta4C3 Nanosheets as a Novel Therapeutic Platform for Photothermal-Driven ROS Scavenging and Immune Activation Against Antibiotic-Resistant Infections in Diabetic Wounds”

IF 13 2区 材料科学 Q1 CHEMISTRY, MULTIDISCIPLINARY
Small Pub Date : 2025-04-25 DOI:10.1002/smll.202502209
Yapeng Wang, Jing Yang, Yunhong Ma, Jun Liu, Peng Wang, Junhao Luo, Yongjun Rui, Yongwei Wu
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引用次数: 0

Abstract

small, 2024, 20, 2400741

DOI: 10.1002/smll.202400741

We sincerely acknowledge an error in the originally published Figure 5i (Ta₄C₃ NSs + NIR group). The correct image is provided below. We confirm that this correction does not affect the scientific conclusions or overall interpretation of the study. We apologize for any confusion this may have caused and appreciate the readers' understanding:

Abstract Image

Figure 5. Assessing Immune Memory Triggered by Ta4C3 NSs and NIR in a Recurrent MRSA Infection Model. A) Illustrates the development of the abscess model and the treatment strategy. On Day 30, the concentrations of serum interleukins IL-4 B), IFN𝛾 C), IL-17 D), and TNF-𝛼 E) were quantified (mean ± SEM, n = 5). F) To evaluate memory B (mB) cells, blood single-cell suspensions were subjected to flow cytometry (FCM) analysis following staining with CD45, CD31, and CD19 antibodies (mean ± SEM, n = 5). G–J) The study included an assessment of CD45+ B220+ memory B cells in lymph nodes (G,H) and spleens (I,J) of mice across different treatment groups on Day 30 (mean ± SEM, n = 5). K,L) The formation of MRSA colonies on LB-agar plates K) and the tally of surviving bacteria L) were documented on day 30. M) Conceptual representation of the mechanism by which Ta4C3 NSs prevent recurrent MRSA infections. Significance levels are marked as ns (not significant), *p < 0.05, **p < 0.01, ***p < 0.001, and ****p < 0.0001, as determined by one-way ANOVA.

We apologize for this error.

更正“Ta4C3纳米片作为光热驱动的ROS清除和免疫激活对抗糖尿病伤口抗生素耐药感染的新治疗平台”
小型,2024,20,24007441 doi: 10.1002/ small . doi: 10.1002。202400741我们真诚地承认最初发表的图5i (Ta₄C₃NSs + NIR组)中的错误。正确的图片如下所示。我们确认此更正不会影响科学结论或研究的整体解释。我们为这可能造成的任何混乱表示歉意,并感谢读者的理解:评估复发性MRSA感染模型中Ta4C3 NSs和NIR触发的免疫记忆。A)说明脓肿模型的发展和治疗策略。30天,血清白细胞介素的浓度il - 4 B),干扰素𝛾C), IL-17 D)和肿瘤坏死因子-𝛼E)量化(±SEM, n = 5)。F)评估记忆B细胞(mB),血液单细胞悬浮液受到流式细胞仪(FCM)分析与CD45染色后,CD31、CD19抗体(±SEM, n = 5)。G-J)的研究包括评估CD45 + B220 +记忆B细胞在淋巴结(G, H)和脾脏(I, J)的小鼠在不同治疗组30天(±SEM,n = 5). K,L)第30天记录lb -琼脂平板上MRSA菌落的形成情况(K)和存活菌数(L)。M) Ta4C3 NSs预防复发性MRSA感染机制的概念性描述。显著性水平用ns(不显著)、*p <;0.05, **p <;0.01, ***p <;0.001, ****p <;0.0001,由单因素方差分析确定。我们为这个错误道歉。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Small
Small 工程技术-材料科学:综合
CiteScore
17.70
自引率
3.80%
发文量
1830
审稿时长
2.1 months
期刊介绍: Small serves as an exceptional platform for both experimental and theoretical studies in fundamental and applied interdisciplinary research at the nano- and microscale. The journal offers a compelling mix of peer-reviewed Research Articles, Reviews, Perspectives, and Comments. With a remarkable 2022 Journal Impact Factor of 13.3 (Journal Citation Reports from Clarivate Analytics, 2023), Small remains among the top multidisciplinary journals, covering a wide range of topics at the interface of materials science, chemistry, physics, engineering, medicine, and biology. Small's readership includes biochemists, biologists, biomedical scientists, chemists, engineers, information technologists, materials scientists, physicists, and theoreticians alike.
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