{"title":"CD248+ fibroblasts drive cardiac fibrosis by interacting with immune cells","authors":"","doi":"10.1038/s44161-025-00625-1","DOIUrl":null,"url":null,"abstract":"A unique subset of cardiac fibroblasts expressing CD248 orchestrates immune–fibroblast crosstalk, promoting chronic cardiac fibrosis and dysfunction after ischemia–reperfusion injury, with potential implications for therapeutic intervention.","PeriodicalId":74245,"journal":{"name":"Nature cardiovascular research","volume":"4 4","pages":"358-359"},"PeriodicalIF":10.8000,"publicationDate":"2025-03-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Nature cardiovascular research","FirstCategoryId":"1085","ListUrlMain":"https://www.nature.com/articles/s44161-025-00625-1","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"CARDIAC & CARDIOVASCULAR SYSTEMS","Score":null,"Total":0}
引用次数: 0
Abstract
A unique subset of cardiac fibroblasts expressing CD248 orchestrates immune–fibroblast crosstalk, promoting chronic cardiac fibrosis and dysfunction after ischemia–reperfusion injury, with potential implications for therapeutic intervention.