Correction to “Overexpression of lncRNA WWTR1-AS1 Associates With Tumor Aggressiveness and Unfavorable Survival in Head–Neck Squamous Cell Carcinoma”

IF 3 3区生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY

Journal of cellular biochemistry Pub Date : 2025-03-02 DOI:10.1002/jcb.70004

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引用次数: 0

Abstract

J. Li, Z. Li, Y. Wu, et al., “Overexpression of lncRNA WWTR1-AS1 Associates With Tumor Aggressiveness and Unfavorable Survival in Head–Neck Squamous Cell Carcinoma,” Journal of Cellular Biochemistry 120, no. 10 (2019): 18266–18277, https://doi.org/10.1002/jcb.29132.

In the published article, the representative flow cytometry plot in Figure 3C (NC Cal27) was included by mistake during figure preparation. The authors have corrected this issue by replacing this image with the correct original flow cytometry plot. The corrected Figure 3 is shown below.

The authors also found that the GAPDH blots in Figure 4D were inadvertently duplicated (Cal27 and FaDu cells treated with either control-ASO or WWTR1-AS1-ASO-393). Based on the original blots, the authors confirm that the GAPDH blot for FaDu cells is correct, while the blot for Cal27 cells has been included by mistake. The corrected Figure 4 is shown below.

The authors apologize for these errors and for any inconvenience these may have caused.

Abstract Image

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更正“lncRNA WWTR1-AS1过表达与头颈部鳞状细胞癌的肿瘤侵袭性和不利生存相关”

李建军，李振华，吴艳，等，“lncRNA WWTR1-AS1在头颈部鳞状细胞癌中的表达与肿瘤侵袭性和不良生存的关系”，《细胞生物化学杂志》，第12期。10 (2019): 18266-18277, https://doi.org/10.1002/jcb.29132.In在已发表的文章中，图3C （NC Cal27）的代表性流式细胞术图在图制备过程中被误入。作者已经纠正了这个问题，用正确的原始流式细胞术图替换了这个图像。更正后的图3如下所示。作者还发现图4D中的GAPDH斑点无意中被复制（Cal27和FaDu细胞分别用control-ASO或WWTR1-AS1-ASO-393处理）。基于原始的印迹，作者确认FaDu细胞的GAPDH印迹是正确的，而Cal27细胞的印迹是错误的。更正后的图4如下所示。作者对这些错误和可能造成的任何不便表示歉意。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Journal of cellular biochemistry 生物-生化与分子生物学

CiteScore

9.90

自引率

0.00%

发文量

164

审稿时长

1 months

期刊介绍： The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.