Heart Disease in Children: Cardiac Dysrhythmias.

Q3 Medicine
FP essentials Pub Date : 2025-02-01
Craig Barstow, Ryan Flanagan
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引用次数: 0

Abstract

Cardiac dysrhythmias in children can be due to a structural abnormality or an intrinsic defect in the electrical conduction system of the heart. In a child with a structurally normal heart, the mechanisms for dysrhythmias are the same as in adults, although the incidence and prevalence are different. Supraventricular tachycardias (SVTs) originate above the ventricles. In children, the two peak ages of onset for SVT are from before birth through the first year of life, and from ages 6 to 8 years. In most cases, there is spontaneous clinical resolution within the first year. When treatment is necessary, it includes vagal maneuvers, pharmacotherapy, and ablation. Wolff-Parkinson-White syndrome is a type of reentrant SVT that involves an accessory pathway. Long QT syndrome is a prolongation of the QT interval due to a genetic channelopathy. Bradycardia and heart block can be due to maternal autoimmune antibodies or structural heart defects. Ventricular tachyarrhythmias can be due to underlying structural heart disease, cardiomyopathy, or a metabolic derangement and may lead to sudden cardiac death.

儿童心脏病:心律失常。
儿童心律失常可能是由于心脏电传导系统的结构异常或内在缺陷。在心脏结构正常的儿童中,心律失常的机制与成人相同,尽管发病率和患病率不同。室上性心动过速(svt)起源于心室上方。在儿童中,SVT发病的两个高峰年龄是从出生前到一岁,以及从6岁到8岁。在大多数情况下,在第一年内有自发的临床消退。当治疗是必要的,它包括迷走神经操作,药物治疗和消融。Wolff-Parkinson-White综合征是一种涉及副通路的可再入性上室t。长QT综合征是由于遗传性通道病引起的QT间期延长。心动过缓和心脏传导阻滞可由母体自身免疫抗体或结构性心脏缺陷引起。室性心动过速可由潜在的结构性心脏病、心肌病或代谢紊乱引起,并可导致心源性猝死。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
FP essentials
FP essentials Medicine-Medicine (all)
CiteScore
1.00
自引率
0.00%
发文量
58
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