RETRACTION: Role of Mirnas in Lung Cancer

IF 4.5 2区生物学 Q2 CELL BIOLOGY

Journal of Cellular Physiology Pub Date : 2025-01-30 DOI:10.1002/jcp.31519

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引用次数: 0

Abstract

RETRACTION: A. Uddin and S. Chakraborty, “Role of miRNAs in Lung Cancer,” Journal of Cellular Physiology (Early View), https://doi.org/10.1002/jcp.26607.

The above article, published online on 20 April 2018 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Robert Heath; and Wiley Periodicals LLC. The retraction has been agreed upon following an investigation into concerns raised by a third party, which revealed major textual overlap between this article and two previously published articles by different groups of authors elsewhere (Guz et al. 2014; Hubaux 2012).

The authors noted the major textual overlap and revised their manuscript during the proofing stage, yet the wrong version of the manuscript was published in Early View by mistake. The editors and the publisher apologize for this oversight. During the investigation, the authors were able to provide their revised article, however, also the revised version shows high textual and conceptual similarity to the two previously published review articles (Guz et al. 2014; Hubaux 2012). The authors disagree with the retraction.

References

Guz, M., A. Rivero-Müller, E. Okoń, et al. 2014. “Micrornas-Role in Lung Cancer.” Disease Markers 2014, no. 1: 1–13. https://doi.org/10.1155/2014/218169.

Hubaux, R. 2012. “Micrornas as Biomarkers for Clinical Features of Lung Cancer.” Journal of Postgenomics: Drug & Biomarker Development 2, no. 3: 1000108. https://doi.org/10.4172/2153-0769.1000108.

查看原文本刊更多论文

撤回：mirna在肺癌中的作用。

撤回：A. Uddin和S. Chakraborty，“miRNAs在肺癌中的作用”，细胞生理学杂志（早期观点），https://doi.org/10.1002/jcp.26607。上述文章于2018年4月20日在线发表在Wiley在线图书馆（wileyonlinelibrary.com）上，经期刊主编Robert Heath同意撤回；和Wiley期刊有限责任公司。在对第三方提出的问题进行调查后，已经同意撤回这篇文章，这篇文章与其他地方不同作者小组发表的两篇文章之间存在主要的文本重叠(Guz et al. 2014；Hubaux 2012)。作者注意到主要的文本重叠，并在校对阶段修改了他们的手稿，但错误的手稿版本被错误地发表在Early View中。编辑和出版商为这个疏忽道歉。在调查过程中，作者能够提供他们的修改后的文章，然而，修改后的文章与之前发表的两篇综述文章在文本和概念上都有很高的相似性(Guz et al. 2014；Hubaux 2012)。作者不同意撤稿。Guz， M.， A. rivero - m ller， E. okoski等。2014。微rna在肺癌中的作用。疾病标志物2014年第1期。1: 1-13。https://doi.org/10.1155/2014/218169。胡博，R. 2012。微rna作为肺癌临床特征的生物标志物。后基因组学杂志：药物与生物标志物开发2，第2期。3: 1000108。https://doi.org/10.4172/2153 - 0769.1000108。

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来源期刊

Journal of Cellular Physiology 生物-生理学

CiteScore

14.70

自引率

0.00%

发文量

256

审稿时长

1 months

期刊介绍： The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.