RETRACTION: Promotion of Cell Autophagy and Apoptosis in Cervical Cancer by Inhibition of Long Noncoding RNA LINC00511 via Transcription Factor RXRA-regulated PLD1

IF 4.5 2区 生物学 Q2 CELL BIOLOGY
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引用次数: 0

Abstract

RETRACTION: Y. Shi, M. Liu, Y. Huang, J. Zhang, and L. Yin, “Promotion of Cell Autophagy and Apoptosis in Cervical Cancer by Inhibition of Long Noncoding RNA LINC00511 via Transcription Factor RXRA-regulated PLD1,” Journal of Cellular Physiology 235, no. 10 (2020): 6592-6604. https://doi.org/10.1002/jcp.29529.

The above article, published online on 17 February 2020 in Wiley Online Library (wileyonlinelibrary.com), and has been retracted by agreement between the journal Editor-in-Chief, Robert Heath; and Wiley Periodicals LLC. A third party reported that images shared overlapping sections in Figure 5D in this article, and this duplication was confirmed by the publisher. The third party also reported that the tumor measurement ruler used in Figure 5 A is identical to the ruler used in the figures of previously published articles from different authors, each of which describes a different scientific context (Chen, et al. 2019 [https://doi.org/10.1002/jcp.28911]) and (Feng, et al. 2019 [https://doi.org/10.1186/s13287-022-02841-z]). The use of the identical ruler in the figures of different articles from unrelated research groups at different institutions puts in doubt the veracity of the data reported. The authors did not respond to an inquiry by the publisher. The retraction has been agreed on because the evidence of image duplication within this article, as well as unexplained duplication of image elements and equipment with other experiments by different authors, fundamentally compromises the conclusions reported in this article. The authors did not respond to our notice regarding the retraction.

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来源期刊
CiteScore
14.70
自引率
0.00%
发文量
256
审稿时长
1 months
期刊介绍: The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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