Corrigendum to “Defining Early Hematopoietic-Fated Primitive Streak Specification of Human Pluripotent Stem Cells by the Orchestrated Balance of Wnt, Activin, and BMP Signaling”
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引用次数: 0
Abstract
This article corrects the following:
Defining Early Hematopoietic-Fated Primitive Streak Specification of Human Pluripotent Stem Cells by the Orchestrated Balance of Wnt, Activin, and BMP Signaling
Jun Shen, Cuicui Lyu, Yaoyao Zhu, Zicen Feng, Shuo Zhang, Dixie L. Hoyle, Guangzhen Ji, Robert A. Brodsky, Tao Cheng, Zack Z. Wang.
Journal of Cell Physiology
https://doi.org/10.1002/jcp.28272
First published: 10 February 2019
Correction text:
The authors made two inadvertent mistakes in the original figures, as follows:
1. The flow cytometry image in the left panel of the original Figure 2e was inadvertently duplicated from the far-right panel of the original Figure 3d.
2. In the original Figure 1d, a duplication of flow figures, for +BMP4 (B) and +BC, occurred. The +BMP4 (B) panel of Figure 1d was incorrectly selected.
The corrected Figure 1 and Figure 2 are as follows:
This correction does not alter the interpretation of the data or the conclusions presented in the paper. The figure legends remain unchanged.
The authors sincerely apologize for any confusion this error may have caused.
沈军，吕翠翠，朱瑶瑶，冯子增，张朔，Dixie L. Hoyle，吉广珍，Robert A. Brodsky，程涛，Zack Z. Wang。Journal of Cell Physiologyhttps://doi.org/10.1002/jcp.28272First出版日期：2019年2月10日更正文本：作者在原图中犯了两个疏忽错误，如下：原图2e左面板的流式细胞术图像无意中与原图3d.2右面板的流式细胞术图像重复。在原始图1d中，出现了+BMP4 (B)和+BC的重复流图。图1d +BMP4 (B)面板选择错误。更正后的图1和图2如下：这一更正不会改变对数据的解释或论文中提出的结论。图形图例保持不变。作者真诚地为这个错误可能造成的任何混乱道歉。
期刊介绍：
The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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