Correction to “SIX1 Is Upregulated in Gastric Cancer and Regulates Proliferation and Invasion by Targeting the ERK Pathway and Promoting Epithelial-Mesenchymal Transition”

IF 2.8 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
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引用次数: 0

Abstract

Y. Xie, P. Jin, X. Sun, T. Jiao, Y. Zhang, Y. Li, and M. Sun, “SIX1 Is Upregulated in Gastric Cancer and Regulates Proliferation and Invasion by Targeting the ERK Pathway and Promoting Epithelial-Mesenchymal Transition,” Cell Biochemistry and Function 36, no. 8 (2018): 413–419, https://doi.org/10.1002/cbf.3361.

In the legend for Figure 2D of the published article, “HGC-7901” should be corrected to “HGC-27”. Additionally, reference 16 of the published article should be corrected to [1]:

H. Lv, A. Cui, F. Sun, Y. Zhang, Y. Li, L. Li, and Z. Lin, “Sineoculis Homeobox Homolog 1 Protein as an Independent Biomarker for Gastric Adenocarcinoma,” Experimental and Molecular Pathology 97, no. 1 (2014): 74–80, https://doi.org/10.1016/j.yexmp.2014.05.007.

The authors apologize for these errors and for the inconvenience these may have caused.

Reference

[1] H. Lv, A. Cui, F. Sun, Y. Zhang, Y. Li, L. Li, and Z. Lin, “Sineoculis Homeobox Homolog 1 Protein as an Independent Biomarker for Gastric Adenocarcinoma,” Experimental and Molecular Pathology 97, no. 1 (2014): 74–80, https://doi.org/10.1016/j.yexmp.2014.05.007.

更正“SIX1在胃癌中上调并通过靶向ERK通路促进上皮-间质转化调控增殖和侵袭”
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来源期刊
Cell Biochemistry and Function
Cell Biochemistry and Function 生物-生化与分子生物学
CiteScore
6.20
自引率
0.00%
发文量
93
审稿时长
6-12 weeks
期刊介绍: Cell Biochemistry and Function publishes original research articles and reviews on the mechanisms whereby molecular and biochemical processes control cellular activity with a particular emphasis on the integration of molecular and cell biology, biochemistry and physiology in the regulation of tissue function in health and disease. The primary remit of the journal is on mammalian biology both in vivo and in vitro but studies of cells in situ are especially encouraged. Observational and pathological studies will be considered providing they include a rational discussion of the possible molecular and biochemical mechanisms behind them and the immediate impact of these observations to our understanding of mammalian biology.
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