RETRACTION: MiR-145 Promotes MiR-133b Expression Through c-myc and DNMT3A-mediated Methylation in Ovarian Cancer Cells

IF 4.5 2区生物学 Q2 CELL BIOLOGY

Journal of Cellular Physiology Pub Date : 2025-01-13 DOI:10.1002/jcp.31507

引用次数: 0

Abstract

RETRACTION: J. Li, S. Zhang, Y. Zou, L. Wu, M. Pei, and Y. Jiang, “MiR-145 Promotes MiR-133b Expression Through c-myc and DNMT3A-mediated Methylation in Ovarian Cancer Cells,” Journal of Cellular Physiology 235, no. 5 (2020): 4291-4301, https://doi.org/10.1002/jcp.29306.

The above article, published online on 14 October 2019 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Roberth Heath; and Wiley Periodicals LLC. The retraction has been agreed due to concerns raised by third parties on the data presented in the article. Specifically, instances of duplicated image elements were identified in Figures 2 d, 3 d, and 5 g. Therefore, the article is retracted as the editors consider its conclusions to be invalid. The authors have been informed of the decision of retraction.

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回归：MiR-145 通过 c-myc 和 DNMT3A 介导的甲基化促进卵巢癌细胞中 MiR-133b 的表达

引用本文：李洁，张生，邹艳，吴丽，裴明，蒋艳，“MiR-145通过c-myc和dnmt3a介导的甲基化促进MiR-133b在卵巢癌细胞中的表达”，《细胞生理杂志》，第35期。5 (2020): 4291-4301, https://doi.org/10.1002/jcp.29306。上述文章于2019年10月14日在线发表在Wiley在线图书馆（wileyonlinelibrary.com）上，经该期刊主编robert Heath；和Wiley期刊有限责任公司。由于第三方对文章中提供的数据提出了担忧，已经同意撤回。具体来说，在图2d、3d和5g中识别了重复图像元素的实例。因此，由于编辑认为其结论无效，文章被撤回。作者已被告知撤稿的决定。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Journal of Cellular Physiology 生物-生理学

CiteScore

14.70

自引率

0.00%

发文量

256

审稿时长

1 months

期刊介绍： The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.