073 The mechanism of RIPK1 S331 O-GlcNAc glycosylation mediated by O-GlcNAc transferase to promote keratinocyte necroptosis in the pathogenesis of severe drug eruption
{"title":"073 The mechanism of RIPK1 S331 O-GlcNAc glycosylation mediated by O-GlcNAc transferase to promote keratinocyte necroptosis in the pathogenesis of severe drug eruption","authors":"C. Zhang, G. Wang, M. Fu","doi":"10.1016/j.jid.2024.10.079","DOIUrl":null,"url":null,"abstract":"","PeriodicalId":16311,"journal":{"name":"Journal of Investigative Dermatology","volume":"144 12","pages":"Page S241"},"PeriodicalIF":5.7000,"publicationDate":"2024-11-20","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Investigative Dermatology","FirstCategoryId":"3","ListUrlMain":"https://www.sciencedirect.com/science/article/pii/S0022202X24022656","RegionNum":2,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"DERMATOLOGY","Score":null,"Total":0}
期刊介绍:
Journal of Investigative Dermatology (JID) publishes reports describing original research on all aspects of cutaneous biology and skin disease. Topics include biochemistry, biophysics, carcinogenesis, cell regulation, clinical research, development, embryology, epidemiology and other population-based research, extracellular matrix, genetics, immunology, melanocyte biology, microbiology, molecular and cell biology, pathology, percutaneous absorption, pharmacology, photobiology, physiology, skin structure, and wound healing