Correction to “Inflammatory Response and Endothelial Dysfunction in the Hearts of Mice Co-Exposed to SO2, NO2, and PM2.5”

IF 4.4 3区医学 Q2 ENVIRONMENTAL SCIENCES

Environmental Toxicology Pub Date : 2024-10-29 DOI:10.1002/tox.24432

引用次数: 0

Abstract

Zhang, Y., Ji, X., Ku, T. and Sang, N. (2016), Inflammatory response and endothelial dysfunction in the hearts of mice co-exposed to SO₂, NO₂, and PM_2.5 . Environmental Toxicology, 31: 1996–2005. https://doi.org/10.1002/tox.22200.

In Figure 4 of the original paper, there was a misuse of the picture that occurred in the low-dose group (A2), and the corrected Figure 4 is presented here.

Abstract Image

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更正 "共同暴露于二氧化硫、二氧化氮和 PM2.5 的小鼠心脏的炎症反应和内皮功能障碍"。

张艳，纪晓明，顾涛，桑宁（2016），SO2、NO2和PM2.5共同暴露小鼠心脏炎症反应和内皮功能障碍。环境毒理学杂志，31(1):1996-2005。https://doi.org/10.1002/tox.22200。在原论文的图4中，出现了低剂量组（A2）对图片的误用，这里给出了更正后的图4。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Environmental Toxicology 环境科学-毒理学

CiteScore

7.10

自引率

8.90%

发文量

261

审稿时长

4.5 months

期刊介绍： The journal publishes in the areas of toxicity and toxicology of environmental pollutants in air, dust, sediment, soil and water, and natural toxins in the environment.Of particular interest are: Toxic or biologically disruptive impacts of anthropogenic chemicals such as pharmaceuticals, industrial organics, agricultural chemicals, and by-products such as chlorinated compounds from water disinfection and waste incineration; Natural toxins and their impacts; Biotransformation and metabolism of toxigenic compounds, food chains for toxin accumulation or biodegradation; Assays of toxicity, endocrine disruption, mutagenicity, carcinogenicity, ecosystem impact and health hazard; Environmental and public health risk assessment, environmental guidelines, environmental policy for toxicants.