Synergistic cross-kingdom host cell damage between Candida albicans and Enterococcus faecalis

Mario Kapitan, Maria Joanna Niemiec, Nicolas Millet, Philipp Brandt, Md Estiak Khan Chowdhury, Anna Czapka, Ketema Abdissa, Franziska Hoffmann, Anna Lange, Mark Veleba, Sandor Nietzsche, Alexander Sandy Mosig, Bettina Loffler, Mike Marquet, Oliwia Makarewicz, Kimberly A. Kline, Slavena Vylkova, Marc Swidergall, Ilse D. Jacobsen
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Abstract

The fungus Candida albicans and the Gram-positive bacterium Enterococcus faecalis share mucosal niches in the human body. As opportunistic pathogens, both are found to expand population size during dysbiosis, and can cause severe systemic infections in susceptible individuals. Here, we show that the presence of C. albicans results in increased host cell damage by E. faecalis. Furthermore, E. faecalis aggravates oropharyngeal candidiasis in mice. Increased damage is mediated by enterococcal cytolysin, and involves both physical interaction and altered glucose availability. Physical interaction promotes accumulation of bacteria on host cells, facilitating contact of cytolysin with host cells. Glucose depletion by the metabolic activity of the fungus sensitized host cells to cytolysin. This work illustrates how a complex interplay between fungi and bacteria can result in detrimental consequences for the host.
白色念珠菌与粪肠球菌之间跨领域的宿主细胞协同损伤
真菌白色念珠菌和革兰氏阳性菌粪肠球菌共享人体粘膜壁龛。作为机会性病原体,它们都会在菌群失调时扩大种群规模,并能在易感人群中引起严重的全身感染。在这里,我们发现白念珠菌的存在会增加粪肠球菌对宿主细胞的损害。此外,粪肠球菌会加重小鼠口咽念珠菌病。损伤加剧是由肠球菌细胞溶解素介导的,涉及物理相互作用和葡萄糖供应的改变。物理作用会促进细菌在宿主细胞上聚集,从而促进细胞溶解素与宿主细胞的接触。真菌的新陈代谢活动导致葡萄糖耗竭,使宿主细胞对细胞溶解素敏感。这项工作说明了真菌和细菌之间复杂的相互作用如何对宿主造成有害后果。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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