Macrophage targeted graphene oxide nanosystem synergize antibiotic killing and host immune defense for Tuberculosis Therapy

IF 9.1 2区 医学 Q1 PHARMACOLOGY & PHARMACY
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Abstract

Tuberculosis (TB), a deadly disease caused by Mycobacterium tuberculosis (Mtb) infection, remains one of the top killers among infectious diseases worldwide. How to increase targeting effects of current anti-TB chemotherapeutics and enhance anti-TB immunological responses remains a big challenge in TB and drug-resistant TB treatment. Here, mannose functionalized and polyetherimide protected graphene oxide system (GO-PEI-MAN) was designed for macrophage-targeted antibiotic (rifampicin) and autophagy inducer (carbamazepine) delivery to achieve more effective Mtb killings by combining targeted drug killing and host immunological clearance. GO-PEI-MAN system demonstrated selective uptake by in vitro macrophages and ex vivo macrophages from macaques. The endocytosed GO-PEI-MAN system would be transported into lysosomes, where the drug loaded Rif@Car@GO-PEI-MAN system would undergo accelerated drug release in acidic lysosomal conditions. Rif@Car@GO-PEI-MAN could significantly promote autophagy and apoptosis in Mtb infected macrophages, as well as induce anti-bacterial M1 polarization of Mtb infected macrophages to increase anti-bacterial IFN-γ and nitric oxide production. Collectively, Rif@Car@GO-PEI-MAN demonstrated effectively enhanced intracellular Mtb killing effects than rifampicin, carbamazepine or GO-PEI-MAN alone in Mtb infected macrophages, and could significantly reduce mycobacterial burdens in the lung of infected mice with alleviated pathology and inflammation without systemic toxicity. This macrophage targeted nanosystem synergizing increased drug killing efficiency and enhanced host immunological defense may be served as more effective therapeutics against TB and drug-resistant TB.

巨噬细胞靶向氧化石墨烯纳米系统协同抗生素杀灭和宿主免疫防御治疗结核病
结核病(TB)是一种由结核分枝杆菌(Mtb)感染引起的致命疾病,仍然是全球传染病中的头号杀手之一。如何提高现有抗结核化疗药物的靶向效果并增强抗结核免疫反应仍是结核病和耐药性结核病治疗中的一大挑战。本文设计了甘露糖功能化和聚醚酰亚胺保护的氧化石墨烯系统(GO-PEI-MAN),用于巨噬细胞靶向抗生素(利福平)和自噬诱导剂(卡马西平)的递送,通过结合靶向药物杀灭和宿主免疫清除,实现更有效的Mtb杀灭。GO-PEI-MAN 系统被体外巨噬细胞和猕猴体外巨噬细胞选择性吸收。内吞的 GO-PEI-MAN 系统会被转运到溶酶体,在酸性溶酶体条件下,装载药物的 Rif@Car@GO-PEI-MAN 系统会加速药物释放。Rif@Car@GO-PEI-MAN能显著促进Mtb感染巨噬细胞的自噬和凋亡,并诱导Mtb感染巨噬细胞的抗菌M1极化,增加抗菌IFN-γ和一氧化氮的产生。总之,与利福平、卡马西平或 GO-PEI-MAN 相比,Rif@Car@GO-PEI-MAN 能有效增强细胞内 Mtb 感染巨噬细胞的杀灭作用,并能显著降低感染小鼠肺部的分枝杆菌负荷,减轻病理和炎症反应,且无全身毒性。这种以巨噬细胞为靶点的纳米系统既能提高药物杀伤效率,又能增强宿主的免疫防御能力,可作为防治结核病和耐药性结核病的更有效疗法。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Pharmacological research
Pharmacological research 医学-药学
CiteScore
18.70
自引率
3.20%
发文量
491
审稿时长
8 days
期刊介绍: Pharmacological Research publishes cutting-edge articles in biomedical sciences to cover a broad range of topics that move the pharmacological field forward. Pharmacological research publishes articles on molecular, biochemical, translational, and clinical research (including clinical trials); it is proud of its rapid publication of accepted papers that comprises a dedicated, fast acceptance and publication track for high profile articles.
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