Trochlea Dysplasia as the Major Anatomic Risk Factors for Patellofemoral Joint Instability: An Infographic as a Visual Learning Tool.

Angelo V Vasiliadis, Theodore Troupis, Dimosthenis Chrysikos, Dimitrios Chytas, George Noussios
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Abstract

Patellofemoral instability (PFI) is a common condition in children and adolescents, ranging from mild discomfort and mal-tracking to lateral patellar dislocation [1]. A number of anatomical risk factors have been described in the literature, such as trochlear dysplasia, patella alta, excessive tibial tuberosity to trochlear groove (TT-TG) distance, patellar tilt and soft tissue alterations [2]. Among them, TD has been identified as the main anatomical risk factor with the strongest association with PFI [1, 2]. In the study by Dejour et al. [2] radiographically observed TD was identified in 96% of patients with a history of patellofemoral dislocation [1, 2]. Patients with Dejour Type D dysplasia are characterized by a prominent and convex lateral facet with a vertical connection to a hypoplastic medial facet (Figure 1), which provides inadequate tracking of the patella in the trochlea during flexion leading to patella subluxation [2]. Non-operative treatment of lateral patellar dislocation presents a rate of re-dislocation up to 70% within 24 months of the first episode [3]. Sulcus deepening trochleoplasty is indicated for Type B and D dysplasia, in order to improve patella tracking, reduce the rate of re-dislocation and achieve good functional outcomes [2, 4]. Trochlea dysplasia is defined as a shallow or flattened groove with decreased resistance to lateral patellar translation. TD can be assessed on computed tomography (CT) and magnetic resonance imaging (MRI) with the use of overlapping axial images and are able to show perfectly the global shape of the femoral trochlea [2]. The thresholds for identifying trochlear dysplasia are established based on the sulcus angle ≥ 145°, medial/lateral trochlea facet asymmetry < 40%, trochlear depth < 3 mm and lateral trochlear inclination ≤ 11° and therefore it can be used in young adolescents [4, 5]. A deep knowledge of anatomic variations and abnormalities of the patellofemoral joint, which may predispose to PFI, is crucial in order to choose the appropriate treatment for each patient.

踝关节发育不良是导致髌股关节不稳的主要解剖学风险因素:作为可视化学习工具的信息图。
髌骨股骨不稳(PFI)是儿童和青少年的一种常见病,轻则出现轻微不适和跟踪不良,重则导致髌骨外侧脱位[1]。文献中描述了一些解剖学上的危险因素,如髌骨发育不良、髌骨突出、胫骨结节与髌骨沟(TT-TG)距离过大、髌骨倾斜和软组织改变等[2]。其中,TD 被认为是与 PFI 关系最密切的主要解剖学风险因素[1, 2]。在 Dejour 等人的研究中[2],96% 有髌骨股骨脱位病史的患者通过影像学观察发现了 TD [1,2]。Dejour D型发育不良患者的特点是外侧面突出且外凸,与发育不良的内侧面垂直连接(图1),这使得髌骨在屈曲时在跗关节内的跟踪不足,导致髌骨半脱位[2]。髌骨外侧脱位的非手术治疗在首次发作后的 24 个月内再脱位率高达 70%[3]。沟深跗关节成形术适用于B型和D型髌骨发育不良,以改善髌骨跟踪,降低再脱位率,达到良好的功能效果[2, 4]。踝关节发育不良的定义是髌沟变浅或变平,髌骨外侧平移阻力减小。TD可通过计算机断层扫描(CT)和磁共振成像(MRI)的轴向重叠图像进行评估,并能完美显示股骨蹄槽的整体形状[2]。鉴定股骨髁发育不良的阈值是根据沟角≥ 145°、内侧/外侧股骨髁面不对称< 40%、股骨髁深度< 3 mm和外侧股骨髁倾斜度≤ 11°确定的,因此可用于青少年[4, 5]。深入了解髌股关节的解剖变异和异常可能导致 PFI,对于为每位患者选择合适的治疗方法至关重要。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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