RIPK1: Inflamed if you do, inflamed if you don’t

IF 25.5 1区医学 Q1 IMMUNOLOGY

Immunity Pub Date : 2024-07-09 DOI:10.1016/j.immuni.2024.06.002

Nicholas W. Hubbard, Andrew Oberst

引用次数: 0

Abstract

RIPK1 is known as a driver of cell death and inflammation. In this issue of Immunity, Imai et al. and Mannion et al. find that these same processes are also induced by RIPK1 inactivation and highlight the therapeutic potential of RIPK1 elimination.

查看原文本刊更多论文

RIPK1：做就发炎，不做也发炎

众所周知，RIPK1 是细胞死亡和炎症的驱动因素。在本期《免疫》杂志上，Imai 等人和 Mannion 等人发现，RIPK1 失活也会诱发这些相同的过程，并强调了消除 RIPK1 的治疗潜力。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

求助全文

约1分钟内获得全文求助全文

来源期刊

Immunity 医学-免疫学

CiteScore

49.40

自引率

2.20%

发文量

205

审稿时长

6 months

期刊介绍： Immunity is a publication that focuses on publishing significant advancements in research related to immunology. We encourage the submission of studies that offer groundbreaking immunological discoveries, whether at the molecular, cellular, or whole organism level. Topics of interest encompass a wide range, such as cancer, infectious diseases, neuroimmunology, autoimmune diseases, allergies, mucosal immunity, metabolic diseases, and homeostasis.