Correction to “Targeting amphiregulin (AREG) derived from senescent stromal cells diminishes cancer resistance and averts programmed cell death 1 ligand (PD-L1)-mediated immunosuppression”
IF 7.8 1区 医学Q1 Biochemistry, Genetics and Molecular Biology
{"title":"Correction to “Targeting amphiregulin (AREG) derived from senescent stromal cells diminishes cancer resistance and averts programmed cell death 1 ligand (PD-L1)-mediated immunosuppression”","authors":"","doi":"10.1111/acel.14248","DOIUrl":null,"url":null,"abstract":"<p>Xu Q, Long Q, Zhu D, et al. Targeting amphiregulin (AREG) derived from senescent stromal cells diminishes cancer resistance and averts programmed cell death 1 ligand (PD-L1)-mediated immunosuppression. <i>Aging Cell</i>. 2019;18:e13027. https://doi.org/10.1111/acel.13027</p><p>During the data organization and author preparation of this manuscript, there were a couple of errors inadvertently incorporated into the manuscript and not recognized effectively during the proofing stage. We noticed that the following item needs to be appropriately corrected.</p><p>Figure 5h. Representative IHC images of caspase 3 (cleaved) in tumors at the end of therapeutic regimes. The “Placebo-treated AREG mAb” and “MIT-treated Cetuximab” images were mistakenly picked up by authors to organize the original panel. As a necessary effort, the authors have now located the appropriate representative images and corrected this figure. Please refer to the updated Figure 5h.</p><p>All other parts of this article remain intact, valid, and unchanged. The authors sincerely regret the error and would like to apologize for any inconvenience this may have caused. The corrected figure is provided below. We apologize for this error.</p>","PeriodicalId":55543,"journal":{"name":"Aging Cell","volume":"23 7","pages":""},"PeriodicalIF":7.8000,"publicationDate":"2024-06-14","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/acel.14248","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Aging Cell","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1111/acel.14248","RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"Biochemistry, Genetics and Molecular Biology","Score":null,"Total":0}
引用次数: 0
Abstract
Xu Q, Long Q, Zhu D, et al. Targeting amphiregulin (AREG) derived from senescent stromal cells diminishes cancer resistance and averts programmed cell death 1 ligand (PD-L1)-mediated immunosuppression. Aging Cell. 2019;18:e13027. https://doi.org/10.1111/acel.13027
During the data organization and author preparation of this manuscript, there were a couple of errors inadvertently incorporated into the manuscript and not recognized effectively during the proofing stage. We noticed that the following item needs to be appropriately corrected.
Figure 5h. Representative IHC images of caspase 3 (cleaved) in tumors at the end of therapeutic regimes. The “Placebo-treated AREG mAb” and “MIT-treated Cetuximab” images were mistakenly picked up by authors to organize the original panel. As a necessary effort, the authors have now located the appropriate representative images and corrected this figure. Please refer to the updated Figure 5h.
All other parts of this article remain intact, valid, and unchanged. The authors sincerely regret the error and would like to apologize for any inconvenience this may have caused. The corrected figure is provided below. We apologize for this error.
Xu Q, Long Q, Zhu D, et al.靶向来自衰老基质细胞的两性胰岛素(AREG)可减轻癌症抗药性并避免程序性细胞死亡1配体(PD-L1)介导的免疫抑制。Aging Cell.2019;18:e13027。https://doi.org/10.1111/acel.13027During,在本稿件的数据整理和作者准备过程中,有几处错误被不经意地纳入稿件,在校对阶段没有被有效识别。图 5h.治疗方案结束时肿瘤中 caspase 3(裂解)的代表性 IHC 图像。"安慰剂处理的 AREG mAb "和 "MIT 处理的西妥昔单抗 "图片是作者在整理原图时误取的。作为必要的努力,作者现已找到适当的代表图像并更正了此图。请参阅更新后的图 5h。本文的所有其他部分保持完整、有效和不变。作者对这一错误表示诚挚的歉意,并对由此造成的不便深表歉意。更正后的图如下。我们对此错误深表歉意。
期刊介绍:
Aging Cell, an Open Access journal, delves into fundamental aspects of aging biology. It comprehensively explores geroscience, emphasizing research on the mechanisms underlying the aging process and the connections between aging and age-related diseases.