Bidirectional relationship between intraindividual changes in behavioral activation and intraindividual changes in postpartum depressive symptoms: A random intercept cross-lagged panel model.

IF 3.1 Q2 PSYCHIATRY
Ivelisse Huerta, Patricio Cumsille, Alvaro Vergés, Lydia Gómez-Pérez
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Abstract

According to Lewisohn's model of depression, decreases in behavioral activation (BA) occurring after facing a vital stressor may increase the risk of depression. Transition to parenthood is a potentially stressful life event that increases the risks of postpartum depression. We aimed to (a) describe the changes in BA and depressive symptomatology between the prepartum period, 1 month, 3 months, and 6 months postpartum and (b) evaluate the bidirectionality of the relationship between intraindividual changes in BA and intraindividual changes in depressive symptoms longitudinally. Chilean pregnant women (N = 503) completed a battery of questionnaires when they were between 32 and 37 weeks of gestation and 1, 3, and 6 months after delivery. A repeated measures analysis of variance showed that BA significantly decreased from prepartum to 1 month postpartum. A random intercept cross-lagged panel model supported the bidirectional inverse relationship between intraindividual changes in BA and intraindividual changes in depressive symptoms. The effect sizes of these associations were large (βs ranging from -.141 to -.243) according to Orth et al. (2022)'s recommendations for cross-lagged effect benchmarks. This relationship showed robustness when multigroup random intercept cross-lagged panel models were conducted to adjust for several covariates (i.e., marital status, the type of health insurance, type of delivery, primiparous vs. multiparous participants, and pregnancy or delivery complication or newborn health problem). Nonetheless, reporting a previous history of major depression moderated this relationship so that intraindividual decreases in BA more likely led to intraindividual increases in depressive symptoms in people with a history of depression than in people without such a history. We discuss implications for behavioral models of depression. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

行为激活的个体内变化与产后抑郁症状的个体内变化之间的双向关系:随机截距交叉滞后面板模型。
根据 Lewisohn 的抑郁模型,在面临重大压力后,行为激活(BA)的减少可能会增加抑郁的风险。为人父母是一个潜在的生活压力事件,会增加产后抑郁的风险。我们的目的是:(a) 描述产前、产后 1 个月、3 个月和 6 个月期间神经激活和抑郁症状的变化;(b) 纵向评估神经激活的个体内变化与抑郁症状的个体内变化之间的双向关系。智利孕妇(503 人)在妊娠 32 至 37 周、产后 1、3 和 6 个月时填写了一系列问卷。重复测量方差分析显示,从产前到产后 1 个月,BA 明显下降。随机截距交叉滞后面板模型支持 BA 的个体内部变化与抑郁症状的个体内部变化之间的双向反比关系。根据 Orth 等人(2022 年)关于跨滞后效应基准的建议,这些关联的效应大小较大(β 值从-.141 到-.243 不等)。当采用多组随机截距交叉滞后面板模型来调整几个协变量(即婚姻状况、医疗保险类型、分娩类型、初产妇与多胎妊娠参与者、妊娠或分娩并发症或新生儿健康问题)时,这种关系显示出稳健性。然而,报告曾有重度抑郁症病史会调节这种关系,因此,与没有抑郁症病史的人相比,有抑郁症病史的人体内 BA 的减少更有可能导致抑郁症状的增加。我们讨论了抑郁症行为模型的意义。(PsycInfo Database Record (c) 2024 APA, 版权所有)。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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