Abdominal wall necrosis after incisional hernia repair in an obese diabetic patient

Helen Brambila Jorge Pareja, Eduardo Alves Canedo, Elisangela Maria Nicolete Rampazzio, José Francisco Galindo Medina Filho, Larissa Pastori, Dayane Valentim Borges, Armando Carromeu Dias Pioch, Victor Hugo Maioli
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Abstract

Tissue repair is a complex and multifactorial organic process, intrinsically related to the health condition of individuals. Systemic pathologies such as hypothyroidism, Diabetes Mellitus (DM) and advanced age have a direct and indirect impact on the chain of physiological processes that make up tissue proliferation and healing. In patients with these comorbidities, especially when they are associated, the mechanism of tissue healing can be compromised and delay their post-operative recovery. The aim of this article is to report a clinical case of an elderly patient, affected by the aforementioned comorbidities, who underwent surgery for numerous abdominal hernias and evolved unfavorably in the postoperative period, with extensive necrosis of the abdominal wall.
一名肥胖糖尿病患者的切口疝修补术后腹壁坏死
组织修复是一个复杂的多因素有机过程,与个人的健康状况有着内在联系。甲状腺功能减退症、糖尿病(DM)和高龄等全身性疾病会直接或间接地影响构成组织增殖和愈合的一系列生理过程。对于患有这些并发症的患者,尤其是伴有这些并发症的患者,组织愈合的机制可能会受到影响,从而延迟他们的术后恢复。本文旨在报告一例老年患者的临床病例,该患者受到上述合并症的影响,接受了多处腹部疝气手术,术后情况不佳,腹壁大面积坏死。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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